IEMbase 0777: TMEM173-related STING superactivity
Scope
| Field | Value |
|---|---|
| IEMbase ID | 777 |
| Nosology | 16.3.09.01 |
| Nosology code | IEM0034 |
| Gene | TMEM173 |
| External IDs | OMIM:615934; ORPHA:481662 |
| Generated mapping | MAPPED; STING_Associated_Vasculopathy_with_Onset_in_Infancy.yaml |
| Candidate DisMech targets | STING_Associated_Vasculopathy_with_Onset_in_Infancy.yaml |
| Review date | 2026-07-08 |
IEMbase phenotype signal
IEMbase labels this autosomal dominant record as TMEM173-related STING superactivity, with alternate names SAVI and dominant familial chilblain lupus type. The source signal includes recurrent infections, fever, interstitial lung disease, acral violaceous plaques and nodules, chilblain lesions, ulcerative lesions with infarcts and gangrene, nail dystrophy or loss, lymphadenopathy, arthralgia, nasal septum perforation, malar flush, arterial and pulmonary hypertension, B lymphopenia, elevated CRP/ESR, elevated IgG, autoantibodies, and a strong interferon-stimulated gene signature.
DisMech phenotype coverage
STING_Associated_Vasculopathy_with_Onset_in_Infancy.yaml is the correct local
target. It models activating STING1/TMEM173 variants, constitutive cGAS/STING
and type I interferon signaling, endothelial activation and small-vessel
vasculopathy, progressive interstitial lung disease/fibrosis, and JAK inhibitor
therapy. Local phenotypes include interstitial lung disease, skin vasculopathy,
failure to thrive, polyarticular arthritis, livedo reticularis, and nasal
septum perforation.
Concordance and completeness
Judgement: exact disease-level coverage; generated mapping should be accepted.
The disease identity, gene, inheritance, interferonopathy mechanism, ILD, skin vasculopathy, arthritis/arthralgia, and nasal involvement are concordant. DisMech is strong for the central STING mechanism and therapeutic context. IEMbase is more granular for inflammatory and immune-laboratory features and for cutaneous complications: CRP/ESR, IgG, autoantibodies, B lymphopenia, recurrent infections, lymphadenopathy, nail dystrophy/loss, acral plaques, and ulceration/gangrene are not all separately represented in the local phenotype set.
Curation actions
- Keep
STING_Associated_Vasculopathy_with_Onset_in_Infancy.yamlas exact coverage for IEMbase 0777. - Consider adding explicit recurrent infections, B lymphopenia, inflammatory marker elevation, lymphadenopathy, nail dystrophy/loss, and ulcerative gangrenous vasculopathy if supported by existing evidence.
- Preserve TMEM173/STING1 naming equivalence: IEMbase uses TMEM173, while the local HGNC label is STING1.