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IEMbase 0352: POMT1-related muscular dystrophy-dystroglycanopathy

Scope

Field Value
IEMbase ID 352
Nosology 18.2.01.06
Gene POMT1
External IDs OMIM:236670; OMIM:613555; OMIM:609308; ORPHA:86812
Generated mapping UNMAPPED; low candidate Dystroglycanopathy.yaml
Candidate DisMech targets Dystroglycanopathy.yaml#MDDG1/POMT1
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents POMT1-CDG/muscular dystrophy-dystroglycanopathy type A1, type B1, and type C1, an autosomal recessive O-mannosylation disorder. Characteristic rows include buphthalmos, cataract, increased creatine kinase, glaucoma, megalocornea, microphthalmia, pigmentary retinopathy, psychomotor delay, normal sialotransferrins, and Walker-Warburg syndrome.

Additional clinical rows include corpus callosum agenesis on MRI, cerebellar abnormalities, cerebral cortical malformations, cobblestone lissencephaly, dysmorphic features, encephalocele, epilepsy, exophthalmia, fatal evolution before 1 year, hydrocephalus, and muscular dystrophy. Biochemical rows include creatine kinase, matriglycan-specific monoclonal antibody, and sialotransferrins. No treatment rows are present.

DisMech phenotype coverage

The generated UNMAPPED status is a false negative. DisMech has a Dystroglycanopathy file that explicitly covers muscular dystrophy-dystroglycanopathy types A/B/C and the POMT1/MDDG1 subtype. Local mechanism describes defective O-mannosyl glycosylation of alpha-dystroglycan, with POMT1 catalyzing the first O-mannosylation step and producing the full severity spectrum from Walker-Warburg syndrome to limb-girdle muscular dystrophy.

Local coverage includes muscular dystrophy, proximal weakness, neonatal hypotonia, elevated serum CK, cobblestone lissencephaly, intellectual disability, retinal dysplasia, hydrocephalus, seizures, reduced alpha-dystroglycan glycosylation, reduced laminin binding, supportive rehabilitation, genetic counseling, and emerging ribitol/AAV therapeutic context.

Concordance and completeness

Judgement: false negative; resolve to the local dystroglycanopathy POMT1 subtype.

The resources agree on POMT1 identity, autosomal recessive inheritance, O-mannosylation/alpha-dystroglycan biology, Walker-Warburg/type A severe spectrum, muscular dystrophy, elevated CK, cobblestone/cortical brain malformations, hydrocephalus, seizures, ocular involvement, psychomotor delay, and early lethality in the severe end.

Curation actions

  • Map this record to Dystroglycanopathy.yaml, specifically the POMT1/MDDG1 subtype context.
  • Consider future enrichment with buphthalmos, megalocornea, microphthalmia, cataract, glaucoma, pigmentary retinopathy, corpus callosum agenesis, encephalocele, fatal-before-1-year wording, and matriglycan antibody testing after source verification.
  • Treat absent IEMbase treatment rows as incomplete IEMbase coverage rather than a contradiction of local supportive and investigational therapy context.