Skip to content

IEMbase 0702: NDUFB3-related NADH dehydrogenase beta subcomplex subunit 3 deficiency

Scope

Field Value
IEMbase ID 702
Nosology 7.1.15.01
Nosology code IEM0427
Gene NDUFB3
External IDs OMIM:618246 for NDUFB3/MC1DN25; IEMbase source lists OMIM:252010; ORPHA:2609
Generated mapping CANDIDATE to COX18-Related_COX_Deficiency.yaml
Candidate DisMech targets Broad complex I/Leigh context only; no exact NDUFB3 target
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents autosomal recessive NDUFB3-related NADH dehydrogenase beta subcomplex subunit 3 deficiency, also labeled mitochondrial complex I deficiency, nuclear type 25.

The source lists OMIM:252010, while MONDO resolves mitochondrial complex I deficiency nuclear type 25 to OMIM:618246 and NDUFB3. The source identifier appears broad or cross-listed and should be reviewed before downstream use.

Biochemical rows show decreased fibroblast complex I activity and increased plasma lactate through childhood. Clinical rows include developmental delay, encephalopathy, hypotonia, myopathy, and characteristic lactic acidosis.

DisMech phenotype coverage

No exact NDUFB3 or MC1DN25 local target was identified.

Leigh_Syndrome.yaml supplies broad overlap for complex I-related neurologic disease, lactate elevation, hypotonia, developmental impairment, and encephalopathy. It does not model NDUFB3.

The generated COX18-Related_COX_Deficiency.yaml candidate is a complex IV COX2-maturation disorder. It shares the nuclear-type number 25 but belongs to MC4DN25 rather than MC1DN25.

Concordance and completeness

Judgement: true local gap with broad Leigh overlap only.

The IEMbase row is a complex I beta-subcomplex disease with neuromuscular and encephalopathic features. COX18 is a wrong-complex number-collision candidate, and the source OMIM field should not be treated as a gene-specific NDUFB3 identifier without review.

Curation actions

  • Add a dedicated NDUFB3/MC1DN25 target if curated.
  • Reject COX18-related complex IV deficiency as exact coverage.
  • Preserve the source OMIM discrepancy for review before downstream identifier use.
  • Preserve decreased fibroblast complex I activity, increased plasma lactate, developmental delay, encephalopathy, hypotonia, myopathy, and lactic acidosis.