Skip to content

IEMbase 0719: COA3-related cytochrome c oxidase assembly factor 3 deficiency

Scope

Field Value
IEMbase ID 719
Nosology 7.4.17.01
Nosology code IEM1146
Gene COA3
External IDs OMIM:619058; ORPHA:254905
Generated mapping UNMAPPED; weak candidate COA3-Related_COX_Deficiency.yaml
Candidate DisMech targets COA3-Related_COX_Deficiency.yaml is exact local coverage
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents autosomal recessive COA3-related cytochrome c oxidase assembly factor 3 deficiency. The cached rows include exercise intolerance, neuropathy, obesity, developmental delay, and short stature, with the exercise-intolerance, neuropathy, and obesity signal concentrated in adolescent and adult windows.

DisMech phenotype coverage

DisMech has exact local coverage in COA3-Related_COX_Deficiency.yaml. The entry resolves to mitochondrial complex IV deficiency nuclear type 14 (MONDO:0033649) and describes biallelic COA3/CCDC56 loss as an inner-membrane complex IV assembly-factor defect that destabilizes COX1 and stalls early COX assembly.

The local phenotype coverage is strong for peripheral neuropathy, exercise intolerance, obesity, and short stature. It also captures the unusually mild, adult-compatible clinical course and the COA3-COX14 interdependence that are not expressed in the compact IEMbase row set.

Concordance and completeness

Judgement: false negative from the generated mapper. The correct target is COA3-Related_COX_Deficiency.yaml.

Identity, inheritance, gene, complex, assembly mechanism, and core phenotype signal all align. IEMbase adds a developmental-delay row that is not prominent in the local COA3 file, while DisMech is richer for the COX1-coupling assembly mechanism and the mild adult-compatible presentation.

Curation actions

  • Resolve IEMbase 719 to COA3-Related_COX_Deficiency.yaml.
  • Treat the generated UNMAPPED status as stale or overly strict.
  • Consider whether the IEMbase developmental-delay row should prompt review of local phenotype breadth.
  • Preserve local mechanistic detail on COA3/COX14 interdependence and COX1 assembly coupling.