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IEMbase 0679: NDUFAF3-related complex I assembly factor 3 deficiency

Scope

Field Value
IEMbase ID 679
Nosology 7.1.03.01
Nosology code IEM0439
Gene NDUFAF3
External IDs OMIM:618240; ORPHA:70474
Generated mapping CANDIDATE to COX6A2-Related_COX_Deficiency.yaml
Candidate DisMech targets Broad complex I/Leigh context only; no exact NDUFAF3 target
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents autosomal recessive NDUFAF3-related complex I assembly factor 3 deficiency, also labeled mitochondrial complex I deficiency, nuclear type 18.

The cached phenotype signal is severe neonatal/infantile mitochondrial disease: decreased fibroblast complex I activity, characteristic increased plasma lactate, hypotonia, diffuse leukomalacia, respiratory failure, perinatal death, and characteristic optic atrophy.

DisMech phenotype coverage

No exact NDUFAF3 or MC1DN18 local target was identified.

Leigh_Syndrome.yaml and other broad mitochondrial entries provide generic complex I/oxidative phosphorylation context but do not model the NDUFAF3 disease entity or its severe neonatal leukomalacia/perinatal-death phenotype.

The generated COX6A2-Related_COX_Deficiency.yaml candidate is a complex IV structural-subunit disorder and should be rejected as exact coverage.

Concordance and completeness

Judgement: true local gap.

The IEMbase row is compact but specific: early lethal complex I assembly-factor disease with lactate elevation, respiratory failure, optic atrophy, and diffuse leukomalacia. A complex IV myopathy/COX candidate does not cover this record.

Curation actions

  • Add a dedicated NDUFAF3/MC1DN18 target if curated.
  • Reject COX6A2-related complex IV deficiency as exact coverage.
  • Preserve decreased fibroblast complex I activity, increased lactate, diffuse leukomalacia, respiratory failure, perinatal death, hypotonia, and optic atrophy.
  • Use broad Leigh/complex I context only as background.