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IEMbase 0031: MTRR-related methionine synthase reductase deficiency, cblE

Scope

Field Value
IEMbase ID 31
Nosology 21.9.12.01
Gene MTRR
External IDs OMIM:236270
Generated mapping MAPPED by alias_exact:cble
Candidate DisMech targets Inborn_Disorder_of_Cobalamin_Metabolism_and_Transport.yaml#cblE
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents methionine synthase reductase deficiency, cblE type. Characteristic features are megaloblastic anemia and neurologic symptoms. Additional clinical features include developmental delay, failure to thrive, ataxia, cerebral atrophy on MRI, hypertonia or hypotonia, adult myelopathy, nystagmus, psychiatric disturbance, seizures, and impaired vision.

The biochemical signal matches an isolated remethylation defect: elevated urine and plasma homocysteine, low-to-normal methionine, normal plasma and urinary methylmalonic acid, and low CSF/plasma S-adenosylmethionine. Treatments listed are hydroxycobalamin and betaine.

DisMech phenotype coverage

The generated subtype mapping is correct. DisMech includes cblE as an MTRR subtype in Inborn_Disorder_of_Cobalamin_Metabolism_and_Transport.yaml, models the MTRR role in reductive reactivation of methionine synthase, and covers the shared remethylation phenotype: homocystinuria, hyperhomocysteinemia, hypomethioninemia, megaloblastic anemia, neurologic injury, developmental delay, seizures, hypotonia, encephalopathy, failure to thrive, hydroxocobalamin, and betaine.

Concordance and completeness

Judgement: correct subtype mapping and good high-level concordance, with the same subtype-granularity limitation seen for cblG.

IEMbase adds cblE-specific normal methylmalonic acid, low SAM in CSF/plasma, cerebral atrophy, myelopathy, nystagmus, impaired vision, psychiatric disturbance, and treatment rows. DisMech is stronger for pathway mechanism and the broader cobalamin-disorder context.

Curation actions

  • Keep the generated subtype mapping.
  • Consider adding cblE-specific biochemical markers and selected neurologic or imaging findings if evidence supports them.
  • Keep cblE and cblG grouped as isolated remethylation defects, but avoid importing methylmalonic acidemia features from cblC/cblA/cblB.