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IEMbase 0384: MAGT1-related Magnesium transporter 1 deficiency (CDG)

Scope

Field Value
IEMbase ID 384
Nosology 18.1.18.01
Gene MAGT1
External IDs OMIM:300716; OMIM:300853; ORPHA:317476
Generated mapping UNMAPPED; low candidate Glycogen_Storage_Disease_Type_I.yaml
Candidate DisMech targets No exact local target
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents X-linked MAGT1-related magnesium transporter 1 deficiency (CDG), with alternate names MAGT1-CDG and immunodeficiency, X-linked, with magnesium defect, Epstein-Barr virus infection and neoplasia.

Clinical rows include T-cell immunodeficiency, magnesium transport defect, neoplasm, psychomotor delay, and Epstein-Barr virus infection. The biochemical signal is normal-to-increased serum sialotransferrins. There are no treatment rows.

DisMech phenotype coverage

There is no exact local DisMech target for MAGT1 deficiency/XMEN disease. The generated Glycogen_Storage_Disease_Type_I.yaml candidate is a false positive: the local file models G6PC1/SLC37A4 glucose-6-phosphatase-system disease with fasting hypoglycemia, hepatomegaly, nephromegaly, lactic acidosis, hyperlipidemia, hyperuricemia, and in GSD Ib neutropenia/infection risk. That does not capture MAGT1-related magnesium transport, EBV susceptibility, or CDG/XMEN biology.

The local CD27-related_lymphoproliferative_and_immune_disorder.yaml file mentions ITK deficiency and MAGT1/XMEN disease as differential EBV-susceptibility context, but it is not an exact MAGT1 target.

Concordance and completeness

Judgement: true MAGT1/XMEN-CDG gap; reject the GSD I candidate.

The IEMbase record is an X-linked magnesium-transporter and glycosylation disorder with EBV susceptibility and neoplasia risk. The generated candidate is a carbohydrate-storage disorder with a different gene, inheritance, mechanism, and clinical signature.

Curation actions

  • Keep this record unmapped until a MAGT1 deficiency/XMEN target exists.
  • Do not map to Glycogen_Storage_Disease_Type_I.yaml.
  • Preserve the CD27/EBV-susceptibility mention only as differential context.
  • If curated, include X-linked inheritance, T-cell immunodeficiency, EBV infection, neoplasia, magnesium transport defect, psychomotor delay, and sialotransferrin findings as review prompts.