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IEMbase 0673: CPT1C-related autosomal dominant spastic paraplegia type 73

Scope

Field Value
IEMbase ID 673
Nosology 4.1.07.01
Nosology code IEM1165
Gene CPT1C
External IDs OMIM:616282; ORPHA:444099
Generated mapping CANDIDATE to Carnitine_Palmitoyltransferase_II_Deficiency.yaml
Candidate DisMech targets No exact CPT1C/SPG73 target identified
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents autosomal dominant CPT1C-related carnitine palmitoyl-transferase 1C deficiency, also labeled spastic paraplegia type 73.

The clinical signal is adult-onset pyramidal motor disease with spastic paraparesis/paraplegia/tetraplegia, hyperreflexia, inability to walk, muscle weakness, muscle atrophy, possible loss of ambulation, and abnormal evoked potentials. The cached rows also include neonatal seizures and a later possible seizure row, which should be source-reviewed because they sit apart from the adult spastic-paraplegia package.

DisMech phenotype coverage

No exact CPT1C or SPG73 local target was identified. Searches of hereditary spastic paraplegia files and groupings did not find CPT1C, SPG73, spastic paraplegia type 73, OMIM:616282, or ORPHA:444099.

The generated Carnitine_Palmitoyltransferase_II_Deficiency.yaml candidate is a carnitine-palmitoyltransferase false positive. CPT II deficiency is a CPT2 long-chain fatty-acid oxidation disorder, not an autosomal dominant CPT1C spastic paraplegia. Carnitine_Palmitoyltransferase_1A_Deficiency.yaml only mentions CPT1C as a brain CPT1 isoform and in population-genetics context, not as disease-level SPG73 coverage.

Concordance and completeness

Judgement: true local gap.

The relevant IEMbase disease concept is neurologic hereditary spastic paraplegia, not hepatic CPT1A deficiency or myopathic CPT2 deficiency. Existing carnitine palmitoyltransferase files are useful only for name disambiguation.

Curation actions

  • Add a dedicated CPT1C/SPG73 target if this record is selected for curation.
  • Reject CPT2 deficiency as exact coverage.
  • Preserve adult spastic paraplegia, hyperreflexia, weakness, atrophy, loss of ambulation, inability to walk, and abnormal evoked potentials.
  • Source-review the neonatal seizure rows before importing them into a CPT1C disease model.