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IEMbase 0711: MT-ND6-related NADH dehydrogenase core subunit 6 deficiency

Scope

Field Value
IEMbase ID 711
Nosology 6.1.24.01
Nosology code IEM0436
Gene MT-ND6
External IDs OMIM:252010; ORPHA:99718
Generated mapping UNMAPPED; weak generated candidate to Pyruvate_Dehydrogenase_Deficiency.yaml
Candidate DisMech targets Broad complex I/Leigh/MELAS context only; no exact MT-ND6 target
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents maternally inherited MT-ND6-related NADH dehydrogenase core subunit 6 deficiency.

Biochemical rows show decreased fibroblast complex I activity and increased plasma lactate across all age windows. Clinical rows include dystonia, lactic acidosis, childhood-to-adult Leber hereditary optic neuropathy, MELAS-like features across all age windows, and stroke-like episodes. Characteristic rows include epilepsy, Leigh syndrome, and optic atrophy.

DisMech phenotype coverage

No exact MT-ND6 local target was identified.

Leigh_Syndrome.yaml provides broad complex I and Leigh-spectrum context. MELAS_Syndrome.yaml provides mitochondrial-gene MELAS context but is most explicit for MT-ND5, not MT-ND6. No exact local LHON target was identified.

The weak generated Pyruvate_Dehydrogenase_Deficiency.yaml candidate is not an mtDNA complex I subunit disorder.

Concordance and completeness

Judgement: true MT-ND6 local gap with broad syndrome overlap only.

The IEMbase row spans complex I deficiency, lactic acidosis, LHON, MELAS-like stroke-like episodes, epilepsy, Leigh syndrome, and optic atrophy. Current local entries are not complete disease-level coverage for MT-ND6.

Curation actions

  • Add a dedicated MT-ND6 complex I deficiency target if curated.
  • Reject pyruvate dehydrogenase deficiency as exact coverage.
  • Preserve decreased complex I activity, increased lactate, dystonia, lactic acidosis, LHON, MELAS-like features, stroke-like episodes, epilepsy, Leigh syndrome, and optic atrophy.