Skip to content

IEMbase 0175: OXCT1-related SCOT deficiency

Scope

Field Value
IEMbase ID 175
Nosology 4.3.02.01
Gene OXCT1
External IDs OMIM:245050; ORPHA:832
Generated mapping UNMAPPED; best candidate Lipoyl_Transferase_1_Deficiency.yaml
Candidate DisMech targets None valid
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents this as OXCT1-related succinyl-CoA:3-oxoacid CoA transferase deficiency, with alternate label SCOT deficiency. Treatability is marked yes.

The biochemical rows emphasize ketone-body utilization: blood and urinary acetoacetate, plasma and urinary ketones, and urinary 3-hydroxybutyric acid range from normal to markedly increased; urinary C6-C10 dicarboxylic acids can be normal to increased; acylglycine, acylcarnitine, serum free carnitine, serum free fatty acids, ammonia, and lactate are represented as normal; and glucose is decreased to normal. Clinical rows include variable cardiomegaly or cardiomyopathy, coma during ketoacidotic episodes, hypoglycemia, acidosis, lethargy, and tachypnea. The treatment row lists avoiding fasting.

DisMech phenotype coverage

No valid local DisMech target was found. The generated best candidate, Lipoyl_Transferase_1_Deficiency.yaml, is a false positive. LIPT1 disease is a lipoylation/mitochondrial dehydrogenase-complex disorder, not an OXCT1 ketone-body utilization disorder. Local search did not find SCOT, OXCT1, or a ketolysis-defect entry.

Concordance and completeness

Judgement: true local gap.

IEMbase describes a distinct ketone-body catabolism disorder with recurrent ketoacidosis and fasting sensitivity. DisMech does not currently have a standalone OXCT1/SCOT deficiency entry.

Curation actions

  • Do not map this record to Lipoyl_Transferase_1_Deficiency.yaml.
  • Add a future OXCT1/SCOT deficiency entry.
  • Expected future coverage: OXCT1, succinyl-CoA:3-oxoacid CoA transferase deficiency, impaired ketone-body utilization, recurrent ketoacidotic crises, acetoacetate/3-hydroxybutyrate/ketone elevation, normal or near-normal ammonia and lactate, hypoglycemia as variable context, cardiomyopathy review, and fasting avoidance/sick-day management.