IEMbase 0756: LIPE-related hormone-sensitive lipase deficiency
Scope
| Field | Value |
|---|---|
| IEMbase ID | 756 |
| Nosology | 14.4.09.01 |
| Nosology code | IEM0663 |
| Gene | LIPE |
| External IDs | OMIM:615980; ORPHA:435660 |
| Generated mapping | UNMAPPED; weak candidate Familial_Partial_Lipodystrophy.yaml |
| Candidate DisMech targets | Familial_Partial_Lipodystrophy.yaml |
| Review date | 2026-07-07 |
IEMbase phenotype signal
IEMbase labels this autosomal recessive record as LIPE-related hormone-sensitive lipase deficiency, with alternate name familial partial lipodystrophy type 6. The source signal includes very high serum cholesterol and triglycerides, elevated creatine kinase, adult lipodystrophy, low body fat percentage, low BMI, diabetes, acanthosis nigricans, and hepatic steatosis.
DisMech phenotype coverage
Familial_Partial_Lipodystrophy.yaml includes a LIPE-related familial partial
lipodystrophy type 6 subtype with biallelic LIPE variant context. The generated
mapper status is therefore a false negative or weak partial hit rather than a
true local absence.
The local FPLD entry covers the main disease-family phenotype cluster: lipodystrophy, insulin resistance or diabetes, hypertriglyceridemia, hepatic steatosis, and acanthosis nigricans. It is less clear for LIPE-specific phenotype detail, particularly creatine kinase elevation, hypercholesterolemia, low BMI, and the adult-predominant onset pattern.
Concordance and completeness
Judgement: false negative with partial local coverage in a broader FPLD entry.
DisMech has the right broad target and subtype identity, but IEMbase adds useful LIPE-specific biochemical and age-pattern prompts that may not be fully represented in the group-level FPLD phenotype set.
Curation actions
- Treat
Familial_Partial_Lipodystrophy.yamlas partial local coverage for LIPE / FPLD6 rather than as an unrelated weak candidate. - Consider adding subtype-specific LIPE phenotype and biochemical detail if the entry supports subtype-level annotations.
- Preserve CK elevation, hypercholesterolemia, low BMI, and adult-onset pattern as source-specific prompts.