IEMbase 0732: COX6B1-related cytochrome c oxidase subunit 6B1 deficiency
Scope
| Field | Value |
|---|---|
| IEMbase ID | 732 |
| Nosology | 7.4.06.02 |
| Nosology code | IEM0467 |
| Gene | COX6B1 |
| External IDs | OMIM:220110; ORPHA:254905 |
| Generated mapping | UNMAPPED; weak candidate COX6B1-Related_COX_Deficiency.yaml |
| Candidate DisMech targets | COX6B1-Related_COX_Deficiency.yaml is exact local coverage |
| Review date | 2026-07-07 |
IEMbase phenotype signal
IEMbase represents autosomal recessive COX6B1-related cytochrome c oxidase subunit 6B1 deficiency. The cached rows include increased plasma lactate across age windows, possible neonatal/infantile/childhood cardiomyopathy and encephalopathy, childhood leukodystrophy, childhood/adolescent myopathy, and possible childhood epilepsy.
DisMech phenotype coverage
DisMech has exact local coverage in COX6B1-Related_COX_Deficiency.yaml. The
entry resolves to mitochondrial complex IV deficiency nuclear type 7
(MONDO:0033637) and describes biallelic COX6B1 variants as loss of a
nuclear-encoded structural subunit of complex IV.
Local phenotype coverage includes severe infantile encephalomyopathy, hydrocephalus, and hypertrophic cardiomyopathy, with the structural-subunit mechanism and reduced COX activity captured in detail.
Concordance and completeness
Judgement: false negative from the generated mapper. The correct target is
COX6B1-Related_COX_Deficiency.yaml.
The IEMbase and local records align on COX6B1, autosomal recessive complex IV structural-subunit disease, encephalopathy, and cardiomyopathy. IEMbase adds age-banded lactate, leukodystrophy, myopathy, and epilepsy prompts, while DisMech adds hydrocephalus and stronger mechanism/evidence context.
Curation actions
- Resolve IEMbase 732 to
COX6B1-Related_COX_Deficiency.yaml. - Treat the generated UNMAPPED status as stale or overly strict.
- Consider reviewing local COX6B1 phenotypes for lactate elevation, leukodystrophy, myopathy, and epilepsy.
- Preserve local hydrocephalus and structural-subunit mechanism detail.