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IEMbase 0768: SLCO2A1-related prostaglandin transporter deficiency

Scope

Field Value
IEMbase ID 768
Nosology 14.3.03.01
Nosology code IEM0686
Gene SLCO2A1
External IDs OMIM:259100; OMIM:119900; ORPHA:468641
Generated mapping CANDIDATE; Primary_Hypertrophic_Osteoarthropathy.yaml
Candidate DisMech targets Primary_Hypertrophic_Osteoarthropathy.yaml subtype PHOAR2
Review date 2026-07-08

IEMbase phenotype signal

IEMbase labels this autosomal recessive record as SLCO2A1-related prostaglandin transporter deficiency, with alternate name primary hypertrophic osteoarthropathy type 2 and abbreviation PHOAR2. The source signal includes high urinary prostaglandin E2 and high urinary prostaglandin M across all age bands, digital clubbing, pachydermia, periostitis, arthralgia, arthritis, swollen joints, thickened skin, hand/foot enlargement, coarse facial features, hyperhidrosis, anemia, myelofibrosis, chronic gastritis, and peptic ulcer.

DisMech phenotype coverage

Primary_Hypertrophic_Osteoarthropathy.yaml is the correct local target. It includes the PHOAR2 subtype for biallelic SLCO2A1 loss-of-function, impaired PGE2 transport, elevated urinary PGE2, elevated urinary PGE-M, periosteal new bone formation, connective tissue proliferation, myelofibrosis, and phenotype coverage for digital clubbing, periostosis, pachydermia, cutis verticis gyrata, arthralgia, hyperhidrosis, joint swelling, anemia, peptic ulcer, and patent ductus arteriosus.

Concordance and completeness

Judgement: exact subtype coverage; generated candidate should be accepted.

The subtype identity, gene, inheritance, prostaglandin transporter mechanism, and biochemical pattern are concordant. IEMbase's PHOAR2-specific phenotype signal usefully emphasizes gastrointestinal and hematologic complications, including chronic gastritis, peptic ulcer, anemia, and myelofibrosis.

Curation actions

  • Treat Primary_Hypertrophic_Osteoarthropathy.yaml subtype PHOAR2 as exact local coverage.
  • Consider adding explicit chronic gastritis coverage if supported by local evidence, because IEMbase lists it separately from peptic ulcer.
  • Preserve the high PGE2 plus high PGE-M biochemical distinction from PHOAR1.