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IEMbase 0479: SLC5A2-related sodium-glucose cotransporter 2 deficiency

Scope

Field Value
IEMbase ID 479
Nosology 3.6.08.01
Gene SLC5A2
External IDs OMIM:233100; ORPHA:69076
Generated mapping UNMAPPED; best candidate Glycogen_Storage_Disease_Type_I.yaml
Candidate DisMech targets Familial_Renal_Glucosuria.yaml; rejected lexical candidate Glycogen_Storage_Disease_Type_I.yaml
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents SLC5A2-related sodium-glucose cotransporter 2 deficiency as familial renal glucosuria type 1 / SGLT2 deficiency. It lists autosomal recessive inheritance. The biochemical rows are focused and renal: urinary glucose is increased across ages, plasma glucose is normal, and urinary amino acids are normal to increased. No clinical or treatment rows are recorded.

DisMech phenotype coverage

Familial_Renal_Glucosuria.yaml is the exact local target, even though the generated mapper missed it. The local entry models familial renal glucosuria as persistent isolated glucosuria without hyperglycemia and without generalized Fanconi-type proximal tubulopathy, with a specific SLC5A2-Related subtype. It captures SLC5A2/SGLT2 loss of function, reduced proximal tubular glucose reabsorption, normal blood glucose with glucosuria, and occasional volume depletion / renin-angiotensin-aldosterone activation in high-glucosuria cases.

Concordance and completeness

Judgement: false negative generated mapping; resolve to Familial_Renal_Glucosuria.yaml#SLC5A2-Related.

The GSD I candidate is a lexical/metabolic false positive. GSD I and GSD Ib have fasting hypoglycemia, liver/kidney glycogen-storage disease, and broad secondary metabolic derangements, while SLC5A2 familial renal glucosuria is an isolated renal glucose-reabsorption defect with normal plasma glucose. The main discordance is inheritance: IEMbase simplifies this row as autosomal recessive, whereas the local entry records semidominant/codominant inheritance with incomplete penetrance for SLC5A2-related disease.

Curation actions

  • Treat this as covered by Familial_Renal_Glucosuria.yaml#SLC5A2-Related.
  • Reject Glycogen_Storage_Disease_Type_I.yaml as an exact mapping.
  • Preserve the inheritance discrepancy as a review flag rather than overwriting the local semidominant/incomplete-penetrance model.