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IEMbase 0371: MTTP-related microsomal triglyceride transfer protein deficiency

Scope

Field Value
IEMbase ID 371
Nosology 15.5.11.01
Gene MTTP
External IDs OMIM:200100; OMIM:157147; ORPHA:14
Generated mapping MAPPED; Abetalipoproteinemia.yaml
Candidate DisMech targets Abetalipoproteinemia.yaml
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents MTTP-related microsomal triglyceride transfer protein deficiency, with alternate names abetalipoproteinemia, Bassen-Kornzweig syndrome, and ABL. Inheritance is autosomal recessive, with prevalence text of approximately 1:1,000,000. The cached record has no treatment rows.

Clinical and biochemical rows include acanthocytosis, ataxia, malabsorption, bleeding tendency, failure to thrive, limited deep tendon reflexes, pigmentary retinopathy, spiculated red cells, prothrombin ratio, plasma HDL cholesterol, plasma LDL cholesterol, lipid-laden small-intestinal biopsy, serum triglyceride, plasma Apo B, plasma vitamin A, and plasma vitamin E.

DisMech phenotype coverage

The generated mapping to Abetalipoproteinemia.yaml is correct. Local DisMech models biallelic MTTP pathogenic variants, failed microsomal triglyceride transfer, absent or extremely low apoB-containing lipoproteins, failure of chylomicron and VLDL export, fat malabsorption, fat-soluble vitamin deficiency, acanthocytosis, retinal disease, neurologic complications, coagulopathy, hepatic complications, low LDL-C, low triglycerides, and low Apo B.

Local coverage is stronger for MTTP mechanism, fat-soluble vitamin replacement, low-fat diet, essential fatty acid intake, and broader management. IEMbase is stronger for specimen-level biomarker rows and small-intestinal biopsy wording.

Concordance and completeness

Judgement: correct mapping with high concordance.

The resources agree on MTTP identity, autosomal recessive inheritance, abetalipoproteinemia/Bassen-Kornzweig disease identity, absent or very low apoB-containing lipoproteins, fat malabsorption, acanthocytosis, failure to thrive, retinopathy, neurologic findings, bleeding/coagulation abnormalities, low LDL-C, low triglycerides, and fat-soluble vitamin deficiency.

Curation actions

  • Keep the generated mapping to Abetalipoproteinemia.yaml.
  • Consider future enrichment with small-intestinal lipid-laden biopsy, prothrombin ratio, plasma HDL cholesterol, limited deep tendon reflexes, and spiculated red cell wording after source verification.
  • Do not treat absent IEMbase treatments as negative evidence; local dietary and fat-soluble vitamin management should remain.