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IEMbase 0701: NDUFA13-related NADH dehydrogenase alpha subcomplex subunit 13 deficiency

Scope

Field Value
IEMbase ID 701
Nosology 7.1.27.01
Nosology code IEM1141
Gene NDUFA13
External IDs OMIM:618249; ORPHA:255241
Generated mapping CANDIDATE to TACO1-Related_COX_Deficiency.yaml
Candidate DisMech targets Broad complex I/Leigh context only; no exact NDUFA13 target
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents autosomal recessive NDUFA13-related NADH dehydrogenase alpha subcomplex subunit 13 deficiency, also labeled mitochondrial complex I deficiency, nuclear type 28.

Biochemical rows show decreased fibroblast complex I activity and increased plasma lactate through childhood. Clinical rows include cerebellar atrophy, developmental delay, encephalopathy, feeding difficulties, lactic acidosis, and characteristic hypotonia.

DisMech phenotype coverage

No exact NDUFA13 or MC1DN28 local target was identified.

Leigh_Syndrome.yaml provides broad overlap for complex I-related mitochondrial encephalopathy, lactate elevation, hypotonia, feeding/bulbar difficulties, and developmental impairment. It does not model NDUFA13-specific disease.

The generated TACO1-Related_COX_Deficiency.yaml candidate is a complex IV mitochondrial translation disorder, not a complex I subunit disease.

Concordance and completeness

Judgement: true local gap with broad Leigh overlap only.

The IEMbase phenotype package includes complex I enzyme deficiency, lactate, cerebellar atrophy, feeding difficulty, encephalopathy, developmental delay, and hypotonia. These should not be attributed to TACO1-related complex IV deficiency.

Curation actions

  • Add a dedicated NDUFA13/MC1DN28 target if curated.
  • Reject TACO1-related complex IV deficiency as exact coverage.
  • Preserve decreased fibroblast complex I activity, increased plasma lactate, cerebellar atrophy, developmental delay, encephalopathy, feeding difficulties, lactic acidosis, and hypotonia.