Skip to content

IEMbase 0554: ABCC2-related Dubin-Johnson syndrome

Scope

Field Value
IEMbase ID 554
Nosology 17.2.02.01
Gene ABCC2
External IDs OMIM:237500; OMIM:601107; ORPHA:234
Generated mapping UNMAPPED; best candidate Stevens-Johnson_Syndrome.yaml
Candidate DisMech targets No exact local target
Review date 2026-07-07

IEMbase phenotype signal

IEMbase represents ABCC2-related canalicular bilirubin glucuronide transporter deficiency, with alternate labels Dubin-Johnson syndrome and ABCC2/DJS. The record is autosomal recessive, and treatability is unknown. No treatment rows are listed.

The biochemical rows include positive ABCC2 sequencing, normal clearance of unconjugated bromsulfthalein, normal urinary coproporphyrin I, normal-to-increased conjugated bilirubin, and pigment granules in liver biopsy. Characteristic clinical rows are episodic jaundice and a normal myocardial ischemia row.

DisMech phenotype coverage

No exact local Dubin-Johnson syndrome target was found for ABCC2 or canalicular bilirubin transport. The generated Stevens-Johnson_Syndrome.yaml candidate is a lexical false positive from the name "Johnson"; it is a severe mucocutaneous drug-reaction phenotype, not an inherited bilirubin transporter disorder.

Local porphyria records mention coproporphyrins, but they do not model ABCC2, black liver pigment, or benign conjugated hyperbilirubinemia.

Concordance and completeness

Judgement: true local disease gap; reject the Stevens-Johnson candidate.

IEMbase provides a focused Dubin-Johnson profile with ABCC2 identity, conjugated bilirubin, liver pigment granules, episodic jaundice, and distinguishing normal coproporphyrin I and bromsulfthalein rows. No current local disease file captures this target.

Curation actions

  • Keep this record unmapped until an ABCC2 / Dubin-Johnson syndrome target exists.
  • Do not map to Stevens-Johnson_Syndrome.yaml or porphyria entries.
  • Preserve liver pigment granules, conjugated bilirubin, episodic jaundice, normal coproporphyrin I, normal bromsulfthalein clearance, and the differential normal myocardial-ischemia row as review prompts.