Aliases: IL1F2, IL-1B, IL1-BETA.
Relationship is the entry's genetic[].relationship_type. untyped means the entry has a genetic record for the gene but does not type it; its free-text association is shown instead.
| Disorder | Relationship | Where the gene appears |
|---|---|---|
| Major Depressive Disorder | modifier | |
| Chronic_Obstructive_Pulmonary_Disease |
untyped
Interleukin-1 beta drives inflammatory signaling.
|
|
| Kawasaki Disease |
untyped
IL-1β promotes endothelial injury, inflammation and remodeling
|
mechanism: Immune Response Dysregulation
|
| Multisystem Inflammatory Syndrome in Children (MIS-C) |
untyped
Implicated in cytokine storm pathophysiology
|
mechanism: Cytokine Storm
|
| Deficiency of the Interleukin-1 Receptor Antagonist | no genetic record |
mechanism: Absent Antagonism at the Interleukin-1 Receptor
|
| Ectopic Pregnancy | no genetic record |
mechanism: Ciliated Epithelial Destruction
|
| Leptospirosis | no genetic record |
mechanism: Cytokine and chemokine upregulation; Increased interleukin-1 beta production
|
| Majeed Syndrome | no genetic record |
mechanism: Excess IL-1 beta Production
|
Identity from HGNC (snapshot 2026-10-01). Page layers are described in the gene pages documentation.