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1
Inheritance
3
Pathophys.
3
Phenotypes
4
Pathograph
1
Genes
🏷

Classifications

👪

Inheritance

1
Autosomal recessive inheritance HP:0000007
DEE83 is caused by a homozygous recurrent UGP2 start-loss variant affecting the shorter brain-predominant isoform.
Autosomal recessive inheritance
Show evidence (1 reference)
PMID:31820119 SUPPORT Human Clinical
"Our study identifies a recurrent start codon mutation in UGP2 as a cause of a novel autosomal recessive DEE syndrome."
The study directly establishes autosomal recessive UGP2-related DEE.

Pathophysiology

3
Brain-predominant UGP2 isoform loss
The recurrent start-loss variant abolishes the shorter UGP2 isoform that is predominant in brain, reducing functional UDP-glucose pyrophosphorylase in neural stem cells.
UGP2 hgnc:12527
UTP:glucose-1-phosphate uridylyltransferase activity GO:0003983 ↓ DECREASED
Show evidence (1 reference)
PMID:31820119 SUPPORT In Vitro
"We show that the absence of the shorter isoform leads to a reduction of functional UGP2 enzyme in neural stem cells"
In-vitro neural stem cell experiments support reduced functional UGP2 as the primary defect.
UDP-glucose-dependent metabolic and glycosylation stress
UDP-glucose shortage in brain-lineage cells disrupts glycogen synthesis and protein glycosylation, with unfolded-protein-response activation.
protein N-linked glycosylation GO:0006487 ⚠ ABNORMAL protein folding in endoplasmic reticulum GO:0034975 ⚠ ABNORMAL
Show evidence (1 reference)
PMID:31820119 SUPPORT In Vitro
"reduced synthesis of UDP-glucose, leading to defects in glycogen synthesis and protein glycosylation"
The full text directly links reduced UDP-glucose synthesis to protein glycosylation defects.
Premature neuronal differentiation and epileptic encephalopathy
The brain-restricted isoform loss produces severe intractable epilepsy, severe developmental delay, progressive microcephaly, and visual disturbance.
neuron CL:0000540
Show evidence (1 reference)
PMID:31820119 SUPPORT Human Clinical
"a severe form of intractable epilepsy, severe developmental delay, progressive microcephaly, visual disturbance and similar minor dysmorphisms."
The patient cohort defines the recurrent neurologic phenotype.

Pathograph

Use the checkboxes to hide or show graph categories. Hover nodes for evidence and cross-linked metadata.
Pathograph: causal mechanism network for UGP2-related developmental and epileptic encephalopathy 83 Interactive directed graph showing how pathophysiology mechanisms, phenotypes, genetic factors and variants, experimental models, environmental triggers, and treatments relate through causal and linked edges.

Phenotypes

3
Head and Neck 1
Progressive microcephaly Microcephaly HP:0000252
Course: PROGRESSIVE
Show evidence (1 reference)
PMID:31820119 SUPPORT Human Clinical
"progressive microcephaly"
The abstract directly reports progressive microcephaly.
Nervous System 1
Global developmental delay Global developmental delay HP:0001263
Show evidence (1 reference)
PMID:31820119 SUPPORT Human Clinical
"severe developmental delay, progressive microcephaly, visual disturbance"
The abstract directly reports severe developmental delay.
Other 1
Developmental and epileptic encephalopathy Epileptic encephalopathy HP:0200134
Show evidence (1 reference)
PMID:31820119 SUPPORT Human Clinical
"presenting with a severe form of intractable epilepsy"
The cohort directly reports severe intractable epilepsy.
🧬

Genetic Associations

1
UGP2 (Homozygous isoform-specific start-loss variant)
Gene: UGP2 hgnc:12527 relationship_type: CAUSATIVE variant_origin: GERMLINE
Show evidence (1 reference)
PMID:31820119 SUPPORT Human Clinical
"Whole exome sequencing identified a recurrent, homozygous variant (chr2:64083454A > G) in the essential UDP-glucose pyrophosphorylase ( UGP2) gene in all probands."
The cohort identifies the recurrent homozygous UGP2 variant.
{ }

Source YAML

click to show
name: UGP2-related developmental and epileptic encephalopathy 83
creation_date: "2026-07-06T06:04:18Z"
description: >-
  UGP2-related developmental and epileptic encephalopathy 83 is an autosomal
  recessive isoform-specific UDP-glucose pyrophosphorylase disorder. A recurrent
  homozygous start-loss variant disrupts the shorter UGP2 isoform that is
  predominant in brain while sparing enough long-isoform function for organismal
  viability. Neural stem cells therefore have reduced functional UGP2 enzyme,
  reduced UDP-glucose production, altered glycogen metabolism, abnormal protein
  glycosylation/proteostasis stress, and premature neuronal differentiation,
  producing severe intractable epilepsy, developmental delay, progressive
  microcephaly, and visual disturbance.
category: Mendelian
disease_term:
  preferred_term: developmental and epileptic encephalopathy, 83
  term:
    id: MONDO:0032895
    label: developmental and epileptic encephalopathy, 83
synonyms:
- DEE83
- EIEE83
parents:
- genetic developmental and epileptic encephalopathy
- congenital disorder of glycosylation
classifications:
  icimd_category:
  - classification_value: multiple_glycosylation_pathways
    notes: >-
      Classified as a multiple-glycosylation-pathway disorder because UGP2
      produces UDP-glucose, a donor/precursor for glycogen metabolism and
      protein glycosylation pathways.
inheritance:
- name: Autosomal recessive inheritance
  inheritance_term:
    preferred_term: Autosomal recessive inheritance
    term:
      id: HP:0000007
      label: Autosomal recessive inheritance
  description: >-
    DEE83 is caused by a homozygous recurrent UGP2 start-loss variant affecting
    the shorter brain-predominant isoform.
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Our study identifies a recurrent start codon mutation in UGP2 as a cause
      of a novel autosomal recessive DEE syndrome.
    explanation: >-
      The study directly establishes autosomal recessive UGP2-related DEE.
pathophysiology:
- name: Brain-predominant UGP2 isoform loss
  description: >-
    The recurrent start-loss variant abolishes the shorter UGP2 isoform that is
    predominant in brain, reducing functional UDP-glucose pyrophosphorylase in
    neural stem cells.
  role: trigger
  genes:
  - preferred_term: UGP2
    term:
      id: hgnc:12527
      label: UGP2
  molecular_functions:
  - preferred_term: UTP:glucose-1-phosphate uridylyltransferase activity
    modifier: DECREASED
    term:
      id: GO:0003983
      label: UTP:glucose-1-phosphate uridylyltransferase activity
  chemical_entities:
  - preferred_term: UDP-D-glucose
    modifier: DECREASED
    term:
      id: CHEBI:18066
      label: UDP-D-glucose
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      We show that the absence of the shorter isoform leads to a reduction of
      functional UGP2 enzyme in neural stem cells
    explanation: >-
      In-vitro neural stem cell experiments support reduced functional UGP2 as
      the primary defect.
  downstream:
  - target: UDP-glucose-dependent metabolic and glycosylation stress
    description: >-
      Reduced functional UGP2 lowers UDP-glucose production in neural stem
      cells, affecting glycogen metabolism, glycosylation, and ER proteostasis.
    causal_link_type: DIRECT
    evidence:
    - reference: PMID:31820119
      reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
      supports: SUPPORT
      evidence_source: IN_VITRO
      snippet: >-
        leading to altered glycogen metabolism, upregulated unfolded protein
        response and premature neuronal differentiation
      explanation: >-
        The abstract connects short-isoform loss to metabolic stress and
        premature neuronal differentiation.
- name: UDP-glucose-dependent metabolic and glycosylation stress
  description: >-
    UDP-glucose shortage in brain-lineage cells disrupts glycogen synthesis and
    protein glycosylation, with unfolded-protein-response activation.
  role: central_effector
  biological_processes:
  - preferred_term: protein N-linked glycosylation
    modifier: ABNORMAL
    term:
      id: GO:0006487
      label: protein N-linked glycosylation
  - preferred_term: protein folding in endoplasmic reticulum
    modifier: ABNORMAL
    term:
      id: GO:0034975
      label: protein folding in endoplasmic reticulum
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      reduced synthesis of UDP-glucose, leading to defects in glycogen synthesis
      and protein glycosylation
    explanation: >-
      The full text directly links reduced UDP-glucose synthesis to protein
      glycosylation defects.
  downstream:
  - target: Premature neuronal differentiation and epileptic encephalopathy
    description: >-
      Neural stem cell stress and premature neuronal differentiation impair
      brain development and produce the developmental epileptic encephalopathy
      phenotype.
    causal_link_type: INDIRECT_KNOWN_INTERMEDIATES
    intermediate_mechanisms:
    - neural stem cell unfolded-protein response
    - premature neuronal differentiation
- name: Premature neuronal differentiation and epileptic encephalopathy
  description: >-
    The brain-restricted isoform loss produces severe intractable epilepsy,
    severe developmental delay, progressive microcephaly, and visual
    disturbance.
  role: consequence
  cell_types:
  - preferred_term: neuron
    term:
      id: CL:0000540
      label: neuron
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      a severe form of intractable epilepsy, severe developmental delay,
      progressive microcephaly, visual disturbance and similar minor
      dysmorphisms.
    explanation: >-
      The patient cohort defines the recurrent neurologic phenotype.
phenotypes:
- name: Developmental and epileptic encephalopathy
  description: Therapy-resistant epilepsy with developmental encephalopathy is central to DEE83.
  phenotype_term:
    preferred_term: Epileptic encephalopathy
    term:
      id: HP:0200134
      label: Epileptic encephalopathy
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      presenting with a severe form of intractable epilepsy
    explanation: The cohort directly reports severe intractable epilepsy.
- name: Global developmental delay
  description: Severe developmental delay is a core feature.
  phenotype_term:
    preferred_term: Global developmental delay
    term:
      id: HP:0001263
      label: Global developmental delay
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      severe developmental delay, progressive microcephaly, visual disturbance
    explanation: The abstract directly reports severe developmental delay.
- name: Progressive microcephaly
  description: Progressive microcephaly occurs in the reported syndrome.
  phenotype_term:
    preferred_term: Microcephaly
    term:
      id: HP:0000252
      label: Microcephaly
    clinical_course: PROGRESSIVE
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      progressive microcephaly
    explanation: The abstract directly reports progressive microcephaly.
genetic:
- name: UGP2
  association: Homozygous isoform-specific start-loss variant
  relationship_type: CAUSATIVE
  variant_origin: GERMLINE
  gene_term:
    preferred_term: UGP2
    term:
      id: hgnc:12527
      label: UGP2
  evidence:
  - reference: PMID:31820119
    reference_title: "Loss of UGP2 in brain leads to a severe epileptic encephalopathy, emphasizing that bi-allelic isoform-specific start-loss mutations of essential genes can cause genetic diseases."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Whole exome sequencing identified a recurrent, homozygous variant
      (chr2:64083454A > G) in the essential UDP-glucose pyrophosphorylase (
      UGP2) gene in all probands.
    explanation: The cohort identifies the recurrent homozygous UGP2 variant.