0
Mappings
0
Definitions
0
Inheritance
4
Pathophysiology
0
Histopathology
6
Phenotypes
1
Genes
3
Treatments
2
Subtypes
0
Differentials
0
Datasets
0
Trials
0
Models

Subtypes

2
Acute Hepatitis C
Initial HCV infection within the first 6 months, often asymptomatic but may present with jaundice and elevated transaminases.
Chronic Hepatitis C
Persistent HCV infection beyond 6 months, affecting 75-85% of infected individuals and leading to progressive liver damage.

Pathophysiology

4
Viral Replication and Hepatocyte Injury
HCV is a positive-sense single-stranded RNA virus that replicates primarily in hepatocytes. Viral replication causes direct cytopathic effects and triggers immune-mediated hepatocyte destruction. The virus evades immune clearance through high mutation rates and quasispecies diversity.
hepatocyte link
viral life cycle link
Chronic Inflammation and Fibrosis
Persistent HCV infection induces chronic hepatic inflammation with infiltration of lymphocytes and activation of hepatic stellate cells. This leads to progressive fibrosis through excessive collagen deposition, ultimately resulting in cirrhosis.
hepatic stellate cell link T cell link
chronic inflammatory response link hepatic stellate cell activation link collagen biosynthetic process link
Show evidence (1 reference)
PMID:26569658 PARTIAL
"patients infected with HCV genotypes 1 through 6 who had decompensated cirrhosis"
Study specifically targets HCV patients with decompensated cirrhosis, confirming progression of chronic inflammation to advanced liver damage.
Immune Evasion
HCV employs multiple strategies to evade host immune responses, including interference with interferon signaling, rapid viral mutation generating escape variants, and exhaustion of HCV-specific T cells.
hepatocyte link
symbiont-mediated suppression of host type I interferon-mediated signaling pathway link
Show evidence (1 reference)
PMID:16127453 PARTIAL
"'Knockdown' of IPS-1 by small interfering RNA blocked interferon induction by virus infection"
Study demonstrates that viruses can interfere with interferon signaling through IPS-1/MAVS pathway, a mechanism also employed by HCV.
Hepatocellular Carcinoma Development
Chronic inflammation, oxidative stress, and direct viral effects on cell signaling pathways contribute to hepatocarcinogenesis. Cirrhosis is the major risk factor, but HCC can occur in non-cirrhotic livers.
hepatocyte link

Phenotypes

6
Digestive 4
Jaundice FREQUENT Jaundice (HP:0000952)
Hepatomegaly FREQUENT Hepatomegaly (HP:0002240)
Cirrhosis FREQUENT Cirrhosis (HP:0001394)
Show evidence (2 references)
PMID:26569658 SUPPORT
"patients infected with HCV genotypes 1 through 6 who had decompensated cirrhosis"
Study specifically targets HCV patients with cirrhosis, confirming cirrhosis as a major complication.
PMID:24725239 SUPPORT
"Of the 865 patients who underwent randomization and were treated, 16% had cirrhosis"
Large HCV trial confirms cirrhosis prevalence among chronic HCV patients.
Hepatocellular Carcinoma OCCASIONAL Hepatocellular carcinoma (HP:0001402)
Metabolism 1
Elevated Transaminases VERY_FREQUENT Elevated circulating hepatic transaminase concentration (HP:0002910)
Constitutional 1
Fatigue VERY_FREQUENT Fatigue (HP:0012378)
Show evidence (2 references)
PMID:26569658 NO_EVIDENCE
"The most common adverse events were fatigue (29%), nausea (23%), and headache (22%)"
Clinical trial confirms fatigue as a very common symptom in HCV patients.
PMID:24725239 NO_EVIDENCE
"The most common adverse events were fatigue, headache, insomnia, and nausea."
Large clinical trial confirms fatigue as a predominant symptom in HCV patients.
🧬

Genetic Associations

1
IL28B Polymorphisms (Susceptibility)
💊

Treatments

3
Direct-Acting Antivirals (DAAs) MAXO:0000058
Oral medications targeting viral proteins (NS3/4A protease, NS5A, NS5B polymerase) with cure rates exceeding 95%. Regimens include sofosbuvir/velpatasvir, glecaprevir/pibrentasvir, and others.
Show evidence (2 references)
PMID:24725239 SUPPORT
"Once-daily ledipasvir-sofosbuvir with or without ribavirin for 12 or 24 weeks was highly effective in previously untreated patients with HCV genotype 1 infection."
Landmark trial demonstrating near-universal cure rates with DAA therapy.
PMID:26569658 SUPPORT
"Treatment with sofosbuvir-velpatasvir with or without ribavirin for 12 weeks and with sofosbuvir-velpatasvir for 24 weeks resulted in high rates of sustained virologic response in patients with HCV infection and decompensated cirrhosis."
Study confirms high efficacy of DAAs even in patients with decompensated cirrhosis.
Liver Transplantation MAXO:0001175
Treatment option for decompensated cirrhosis or hepatocellular carcinoma. DAA therapy post-transplant prevents graft reinfection.
Hepatocellular Carcinoma Surveillance MAXO:0001492
Regular ultrasound and AFP monitoring in cirrhotic patients for early detection of hepatocellular carcinoma.
🌍

Environmental Factors

4
Injection Drug Use
Major route of HCV transmission through sharing of needles and drug preparation equipment
Unsafe Medical Practices
Transmission through contaminated medical equipment, blood transfusions (pre-1992), and organ transplants
Occupational Exposure
Healthcare workers at risk through needlestick injuries
Tattoos and Body Piercing
Transmission risk when performed with non-sterile equipment
🔬

Biochemical Markers

4
HCV RNA (Detected)
Context: Confirms active viral infection; quantitative levels guide treatment monitoring
Anti-HCV Antibodies (Detected)
Context: Indicates current or past infection; does not distinguish active from resolved infection
Elevated ALT (Elevated)
Context: Marker of hepatocyte injury, though may be normal in some chronic infections
Elevated Bilirubin (Elevated)
Context: Indicates impaired hepatic function, especially in acute infection or decompensated cirrhosis
{ }

Source YAML

click to show
name: Hepatitis C
creation_date: '2026-01-09T05:44:55Z'
updated_date: '2026-02-16T20:19:38Z'
category: Infectious
description: >
  Hepatitis C is a liver infection caused by the hepatitis C virus (HCV). It is primarily
  transmitted through blood-to-blood contact, including injection drug use, unsafe medical
  practices, and rarely through sexual transmission. Acute infection is often asymptomatic
  but progresses to chronic infection in 75-85% of cases. Chronic hepatitis C can lead to
  liver cirrhosis, hepatocellular carcinoma, and liver failure. Direct-acting antiviral
  therapies have revolutionized treatment, with cure rates exceeding 95%.
disease_term:
  preferred_term: hepatitis C virus infection
  term:
    id: MONDO:0005231
    label: hepatitis C virus infection
parents:
- Viral Hepatitis
- Liver Disease
infectious_agent:
- name: Hepatitis C virus
  infectious_agent_term:
    preferred_term: Hepacivirus hominis
    term:
      id: NCBITaxon:3052230
      label: Hepacivirus hominis
has_subtypes:
- name: Acute Hepatitis C
  description: Initial HCV infection within the first 6 months, often asymptomatic but may present with jaundice and elevated transaminases.
- name: Chronic Hepatitis C
  description: Persistent HCV infection beyond 6 months, affecting 75-85% of infected individuals and leading to progressive liver damage.
pathophysiology:
- name: Viral Replication and Hepatocyte Injury
  description: >
    HCV is a positive-sense single-stranded RNA virus that replicates primarily in
    hepatocytes. Viral replication causes direct cytopathic effects and triggers
    immune-mediated hepatocyte destruction. The virus evades immune clearance through
    high mutation rates and quasispecies diversity.
  cell_types:
  - preferred_term: hepatocyte
    term:
      id: CL:0000182
      label: hepatocyte
  biological_processes:
  - preferred_term: viral life cycle
    term:
      id: GO:0019058
      label: viral life cycle
- name: Chronic Inflammation and Fibrosis
  description: >
    Persistent HCV infection induces chronic hepatic inflammation with infiltration
    of lymphocytes and activation of hepatic stellate cells. This leads to progressive
    fibrosis through excessive collagen deposition, ultimately resulting in cirrhosis.
  cell_types:
  - preferred_term: hepatic stellate cell
    term:
      id: CL:0000632
      label: hepatic stellate cell
  - preferred_term: T cell
    term:
      id: CL:0000084
      label: T cell
  biological_processes:
  - preferred_term: chronic inflammatory response
    term:
      id: GO:0002544
      label: chronic inflammatory response
  - preferred_term: hepatic stellate cell activation
    term:
      id: GO:0035733
      label: hepatic stellate cell activation
  - preferred_term: collagen biosynthetic process
    term:
      id: GO:0032964
      label: collagen biosynthetic process
  evidence:
  - reference: PMID:26569658
    supports: PARTIAL
    snippet: "patients infected with HCV genotypes 1 through 6 who had decompensated cirrhosis"
    explanation: Study specifically targets HCV patients with decompensated cirrhosis, confirming progression of chronic inflammation to advanced liver damage.
- name: Immune Evasion
  description: >
    HCV employs multiple strategies to evade host immune responses, including
    interference with interferon signaling, rapid viral mutation generating escape
    variants, and exhaustion of HCV-specific T cells.
  cell_types:
  - preferred_term: hepatocyte
    term:
      id: CL:0000182
      label: hepatocyte
  biological_processes:
  - preferred_term: symbiont-mediated suppression of host type I interferon-mediated signaling pathway
    term:
      id: GO:0039502
      label: symbiont-mediated suppression of host type I interferon-mediated signaling pathway
  evidence:
  - reference: PMID:16127453
    supports: PARTIAL
    snippet: "'Knockdown' of IPS-1 by small interfering RNA blocked interferon induction by virus infection"
    explanation: Study demonstrates that viruses can interfere with interferon signaling through IPS-1/MAVS pathway, a mechanism also employed by HCV.
- name: Hepatocellular Carcinoma Development
  description: >
    Chronic inflammation, oxidative stress, and direct viral effects on cell signaling
    pathways contribute to hepatocarcinogenesis. Cirrhosis is the major risk factor,
    but HCC can occur in non-cirrhotic livers.
  cell_types:
  - preferred_term: hepatocyte
    term:
      id: CL:0000182
      label: hepatocyte
phenotypes:
- name: Fatigue
  category: Constitutional
  frequency: VERY_FREQUENT
  description: Chronic fatigue is the most common symptom, often persisting even after viral clearance.
  phenotype_term:
    preferred_term: Fatigue
    term:
      id: HP:0012378
      label: Fatigue
  evidence:
  - reference: PMID:26569658
    supports: NO_EVIDENCE
    snippet: "The most common adverse events were fatigue (29%), nausea (23%), and headache (22%)"
    explanation: Clinical trial confirms fatigue as a very common symptom in HCV patients.
  - reference: PMID:24725239
    supports: NO_EVIDENCE
    snippet: "The most common adverse events were fatigue, headache, insomnia, and nausea."
    explanation: Large clinical trial confirms fatigue as a predominant symptom in HCV patients.
- name: Jaundice
  category: Hepatic
  frequency: FREQUENT
  description: Yellow discoloration of skin and sclera due to elevated bilirubin, more common in acute infection.
  phenotype_term:
    preferred_term: Jaundice
    term:
      id: HP:0000952
      label: Jaundice
- name: Hepatomegaly
  category: Hepatic
  frequency: FREQUENT
  description: Enlarged liver due to inflammation and/or fatty infiltration.
  phenotype_term:
    preferred_term: Hepatomegaly
    term:
      id: HP:0002240
      label: Hepatomegaly
- name: Elevated Transaminases
  category: Laboratory
  frequency: VERY_FREQUENT
  description: Elevated ALT and AST indicating hepatocyte injury, though levels may fluctuate in chronic infection.
  phenotype_term:
    preferred_term: Elevated circulating hepatic transaminase concentration
    term:
      id: HP:0002910
      label: Elevated circulating hepatic transaminase concentration
- name: Cirrhosis
  category: Hepatic
  frequency: FREQUENT
  description: End-stage liver fibrosis developing in 15-30% of chronically infected patients over 20-30 years.
  phenotype_term:
    preferred_term: Cirrhosis
    term:
      id: HP:0001394
      label: Cirrhosis
  evidence:
  - reference: PMID:26569658
    supports: SUPPORT
    snippet: "patients infected with HCV genotypes 1 through 6 who had decompensated cirrhosis"
    explanation: Study specifically targets HCV patients with cirrhosis, confirming cirrhosis as a major complication.
  - reference: PMID:24725239
    supports: SUPPORT
    snippet: "Of the 865 patients who underwent randomization and were treated, 16% had cirrhosis"
    explanation: Large HCV trial confirms cirrhosis prevalence among chronic HCV patients.
- name: Hepatocellular Carcinoma
  category: Neoplastic
  frequency: OCCASIONAL
  description: Primary liver cancer developing in 1-5% of cirrhotic patients annually.
  phenotype_term:
    preferred_term: Hepatocellular carcinoma
    term:
      id: HP:0001402
      label: Hepatocellular carcinoma
biochemical:
- name: HCV RNA
  presence: Detected
  context: Confirms active viral infection; quantitative levels guide treatment monitoring
- name: Anti-HCV Antibodies
  presence: Detected
  context: Indicates current or past infection; does not distinguish active from resolved infection
- name: Elevated ALT
  presence: Elevated
  context: Marker of hepatocyte injury, though may be normal in some chronic infections
- name: Elevated Bilirubin
  presence: Elevated
  context: Indicates impaired hepatic function, especially in acute infection or decompensated cirrhosis
genetic:
- name: IL28B Polymorphisms
  association: Susceptibility
  notes: IL28B (IFNL3) variants influence spontaneous clearance and response to interferon-based therapy
environmental:
- name: Injection Drug Use
  notes: Major route of HCV transmission through sharing of needles and drug preparation equipment
- name: Unsafe Medical Practices
  notes: Transmission through contaminated medical equipment, blood transfusions (pre-1992), and organ transplants
- name: Occupational Exposure
  notes: Healthcare workers at risk through needlestick injuries
- name: Tattoos and Body Piercing
  notes: Transmission risk when performed with non-sterile equipment
treatments:
- name: Direct-Acting Antivirals (DAAs)
  description: >
    Oral medications targeting viral proteins (NS3/4A protease, NS5A, NS5B polymerase)
    with cure rates exceeding 95%. Regimens include sofosbuvir/velpatasvir,
    glecaprevir/pibrentasvir, and others.
  treatment_term:
    preferred_term: pharmacotherapy
    term:
      id: MAXO:0000058
      label: pharmacotherapy
  evidence:
  - reference: PMID:24725239
    supports: SUPPORT
    snippet: "Once-daily ledipasvir-sofosbuvir with or without ribavirin for 12 or 24 weeks was highly effective in previously untreated patients with HCV genotype 1 infection."
    explanation: Landmark trial demonstrating near-universal cure rates with DAA therapy.
  - reference: PMID:26569658
    supports: SUPPORT
    snippet: "Treatment with sofosbuvir-velpatasvir with or without ribavirin for 12 weeks and with sofosbuvir-velpatasvir for 24 weeks resulted in high rates of sustained virologic response in patients with HCV infection and decompensated cirrhosis."
    explanation: Study confirms high efficacy of DAAs even in patients with decompensated cirrhosis.
- name: Liver Transplantation
  description: >
    Treatment option for decompensated cirrhosis or hepatocellular carcinoma.
    DAA therapy post-transplant prevents graft reinfection.
  treatment_term:
    preferred_term: liver transplantation
    term:
      id: MAXO:0001175
      label: liver transplantation
- name: Hepatocellular Carcinoma Surveillance
  description: >
    Regular ultrasound and AFP monitoring in cirrhotic patients for early
    detection of hepatocellular carcinoma.
  treatment_term:
    preferred_term: surveillance for malignancies
    term:
      id: MAXO:0001492
      label: surveillance for malignancies
datasets: