0
Mappings
0
Definitions
0
Inheritance
5
Pathophysiology
1
Histopathology
8
Phenotypes
5
Pathograph
4
Genes
5
Treatments
2
Subtypes
0
Differentials
0
Datasets
0
Trials
0
Models
🏷

Classifications

Harrison's Chapter
cancer hematologic malignancy
ICD-O Morphology
Leukemia

Subtypes

2
IGHV Mutated CLL
CLL with somatic hypermutation of immunoglobulin heavy chain variable region genes (at least 2% deviation from germline). Associated with favorable prognosis, lower risk of progression, and better responses to chemoimmunotherapy. Represents post-germinal center B-cell origin.
IGHV Unmutated CLL
CLL without significant IGHV somatic hypermutation (less than 2% deviation from germline). Associated with more aggressive course, higher risk of progression, and inferior outcomes with chemoimmunotherapy. May benefit particularly from novel targeted agents.

Pathophysiology

5
B-cell Receptor Signaling Dysregulation
CLL cells exhibit constitutive B-cell receptor (BCR) signaling, either through autonomous signaling or antigen-driven activation. This chronic BCR stimulation activates downstream kinases including BTK, PI3K, and SYK, driving proliferation and survival. BCR signaling intensity correlates with clinical aggressiveness.
mature B cell link
B cell activation link ↑ INCREASED
BTK Pathway Activation
Bruton tyrosine kinase (BTK) is essential for BCR signaling and CLL cell survival. BTK activation promotes NF-kB signaling, cell adhesion, and tissue homing. BTK inhibitors disrupt these signals, mobilizing CLL cells from protective tissue microenvironments and inducing apoptosis.
signal transduction link ↑ INCREASED
Microenvironmental Survival Signals
CLL cells depend on interactions with the tissue microenvironment including nurse-like cells, T cells, and stromal cells in lymph nodes and bone marrow. These interactions provide survival signals through chemokines, cytokines, and direct cell contact. Disrupting these interactions is therapeutically important.
B cell link
lymph node link bone marrow link
NF-kB Activation and Survival
NF-kB transcription factors are constitutively activated in CLL and promote expression of anti-apoptotic genes including BCL2, MCL1, and XIAP. This contributes to the apoptosis resistance characteristic of CLL cells.
apoptotic process link ↓ DECREASED
CLL Cell Accumulation
The combination of defective apoptosis, microenvironmental support, and low-level proliferation leads to progressive accumulation of clonal B cells. CLL is primarily a disease of accumulation rather than rapid proliferation.
B cell link

Histopathology

1
Small B-Cell Clonal Expansion VERY_FREQUENT
Chronic lymphocytic leukemia is characterized by clonal expansion of CLL B cells.
Show evidence (1 reference)
PMID:29670635 SUPPORT
"Chronic lymphocytic leukemia (CLL) is characterized by the clonal expansion of"
Abstract describes CLL as characterized by clonal expansion of CLL B cells.

Pathograph

Use the checkboxes to hide or show graph categories. Hover nodes for evidence and cross-linked metadata.
Pathograph: causal mechanism network for Chronic Lymphocytic Leukemia Interactive directed graph showing how pathophysiology mechanisms, phenotypes, genetic factors and variants, experimental models, environmental triggers, and treatments relate through causal and linked edges.

Phenotypes

8
Blood 3
Lymphoproliferative Disorder VERY_FREQUENT Lymphoproliferative disorder (HP:0005523)
Anemia FREQUENT Anemia (HP:0001903)
Thrombocytopenia FREQUENT Thrombocytopenia (HP:0001873)
Cardiovascular 2
Lymphadenopathy VERY_FREQUENT Lymphadenopathy (HP:0002716)
Show evidence (1 reference)
PMID:1139039 SUPPORT Human Clinical
"stage 1, lymphocytosis with enlarged nodes"
The Rai staging system defines Stage I CLL by the presence of lymphadenopathy (enlarged nodes), confirming its clinical significance.
Splenomegaly FREQUENT Splenomegaly (HP:0001744)
Show evidence (1 reference)
PMID:1139039 SUPPORT Human Clinical
"stage II, lymphocytosis with enlarged spleen or liver or both"
The Rai staging system defines Stage II CLL by the presence of splenomegaly or hepatomegaly, confirming splenomegaly as a key clinical feature.
Immune 1
Recurrent Infections FREQUENT Recurrent infections (HP:0002719)
Show evidence (1 reference)
PMID:18755702 SUPPORT Human Clinical
"Patients with chronic lymphocytic leukaemia (CLL) have progressive immunodeficiency and infection is the commonest cause of death."
This authoritative review establishes that infection due to immunodeficiency is the leading cause of death in CLL patients.
Constitutional 1
Night Sweats OCCASIONAL Night sweats (HP:0030166)
Growth 1
Weight Loss OCCASIONAL Weight loss (HP:0001824)
🧬

Genetic Associations

4
TP53 Mutation/del(17p) (High-Risk Prognostic Marker)
del(11q)/ATM (Intermediate-Risk Marker)
del(13q) (Favorable Prognostic Marker)
IGHV (Prognostic Marker)
💊

Treatments

5
Ibrutinib
Action: pharmacotherapy MAXO:0000058
Agent: ibrutinib
First-in-class irreversible BTK inhibitor that disrupts BCR signaling, mobilizes CLL cells from tissue niches, and induces apoptosis. Effective in all risk groups including TP53-mutated CLL. Continuous therapy until progression.
Show evidence (1 reference)
PMID:26639149 SUPPORT Human Clinical
"ibrutinib resulted in significantly longer progression-free survival than did chlorambucil (median, not reached vs. 18.9 months), with a risk of progression or death that was 84% lower with ibrutinib than that with chlorambucil"
The landmark RESONATE-2 trial demonstrated ibrutinib's superiority in treatment-naive elderly CLL patients with 84% reduction in risk of progression or death.
Acalabrutinib
Action: pharmacotherapy MAXO:0000058
Agent: acalabrutinib
Second-generation selective BTK inhibitor with improved kinase selectivity and potentially fewer off-target effects than ibrutinib. Effective as monotherapy or combined with obinutuzumab.
Venetoclax plus Obinutuzumab
Action: pharmacotherapy MAXO:0000058
Agent: venetoclax
BCL2 inhibitor venetoclax combined with anti-CD20 antibody obinutuzumab achieves high rates of undetectable MRD and enables time-limited therapy (12 months). Particularly effective in treatment-naive CLL.
Show evidence (1 reference)
PMID:30523712 SUPPORT Human Clinical
"The MURANO study demonstrated significant progression-free survival (PFS) benefit for fixed-duration venetoclax-rituximab compared with bendamustine-rituximab in relapsed/refractory chronic lymphocytic leukemia."
The MURANO Phase III study demonstrated venetoclax-rituximab's superiority in relapsed/refractory CLL with significantly improved progression-free survival.
Chemoimmunotherapy (FCR)
Action: chemotherapy MAXO:0000647
Agent: cyclophosphamide
Fludarabine, cyclophosphamide, and rituximab combination was standard for fit patients with mutated IGHV without del(17p)/TP53 mutation. Can achieve long remissions in this favorable subgroup but largely replaced by targeted agents.
Watch and Wait
Action: surveillance for malignancies MAXO:0001492
Asymptomatic early-stage CLL does not require immediate treatment. Active surveillance with monitoring for disease progression or treatment indications (cytopenias, symptomatic disease, rapid progression) is appropriate for many patients.
🔬

Biochemical Markers

2
Flow Cytometry Immunophenotyping
IGHV Mutation Analysis
{ }

Source YAML

click to show
name: Chronic Lymphocytic Leukemia
creation_date: '2026-01-26T02:55:13Z'
updated_date: '2026-03-13T12:00:00Z'
description: >-
  Chronic lymphocytic leukemia (CLL) is the most common leukemia in Western countries,
  characterized by clonal expansion of mature-appearing CD5+ B lymphocytes in blood,
  bone marrow, and lymphoid tissues. The clinical course is highly variable, ranging
  from indolent disease requiring no treatment to aggressive forms. Prognosis is
  stratified by IGHV mutation status, cytogenetics (del(17p), del(11q), del(13q)),
  and TP53 mutations. Treatment has been revolutionized by BTK inhibitors (ibrutinib,
  acalabrutinib) and BCL2 inhibitors (venetoclax), enabling chemotherapy-free regimens
  with deep remissions.
categories:
- Hematologic Malignancy
- B-cell Neoplasm
- Chronic Leukemia
- Lymphoproliferative Disorder
parents:
- B-cell neoplasm
epidemiology:
- name: Most common adult leukemia
  description: Chronic lymphocytic leukemia is the most common type of leukemia in adults.
  evidence:
  - reference: PMID:41572562
    reference_title: "\"Leukemic pernio\": perniosis-like presentations of chronic lymphocytic leukemia."
    supports: SUPPORT
    snippet: "Chronic lymphocytic leukemia (CLL) is the most common type of leukemia in adults."
    explanation: This abstract explicitly states that CLL is the most common adult leukemia.
has_subtypes:
- name: IGHV Mutated CLL
  description: >-
    CLL with somatic hypermutation of immunoglobulin heavy chain variable region
    genes (at least 2% deviation from germline). Associated with favorable prognosis,
    lower risk of progression, and better responses to chemoimmunotherapy. Represents
    post-germinal center B-cell origin.
- name: IGHV Unmutated CLL
  description: >-
    CLL without significant IGHV somatic hypermutation (less than 2% deviation from
    germline). Associated with more aggressive course, higher risk of progression,
    and inferior outcomes with chemoimmunotherapy. May benefit particularly from
    novel targeted agents.
pathophysiology:
- name: B-cell Receptor Signaling Dysregulation
  description: >-
    CLL cells exhibit constitutive B-cell receptor (BCR) signaling, either through
    autonomous signaling or antigen-driven activation. This chronic BCR stimulation
    activates downstream kinases including BTK, PI3K, and SYK, driving proliferation
    and survival. BCR signaling intensity correlates with clinical aggressiveness.
  cell_types:
  - preferred_term: mature B cell
    term:
      id: CL:0000785
      label: mature B cell
  biological_processes:
  - preferred_term: B cell activation
    modifier: INCREASED
    term:
      id: GO:0042113
      label: B cell activation
  downstream:
  - target: BTK Pathway Activation
    description: BCR engagement activates BTK-mediated survival signaling
  - target: Microenvironmental Survival Signals
    description: BCR signaling facilitates interaction with supportive niches
- name: BTK Pathway Activation
  description: >-
    Bruton tyrosine kinase (BTK) is essential for BCR signaling and CLL cell survival.
    BTK activation promotes NF-kB signaling, cell adhesion, and tissue homing.
    BTK inhibitors disrupt these signals, mobilizing CLL cells from protective
    tissue microenvironments and inducing apoptosis.
  biological_processes:
  - preferred_term: signal transduction
    modifier: INCREASED
    term:
      id: GO:0007165
      label: signal transduction
  downstream:
  - target: NF-kB Activation and Survival
    description: BTK activates NF-kB-mediated anti-apoptotic gene expression
- name: Microenvironmental Survival Signals
  description: >-
    CLL cells depend on interactions with the tissue microenvironment including
    nurse-like cells, T cells, and stromal cells in lymph nodes and bone marrow.
    These interactions provide survival signals through chemokines, cytokines,
    and direct cell contact. Disrupting these interactions is therapeutically
    important.
  locations:
  - preferred_term: lymph node
    term:
      id: UBERON:0000029
      label: lymph node
  - preferred_term: bone marrow
    term:
      id: UBERON:0002371
      label: bone marrow
  cell_types:
  - preferred_term: B cell
    term:
      id: CL:0000236
      label: B cell
  downstream:
  - target: CLL Cell Accumulation
    description: Microenvironmental support enables CLL expansion
- name: NF-kB Activation and Survival
  description: >-
    NF-kB transcription factors are constitutively activated in CLL and promote
    expression of anti-apoptotic genes including BCL2, MCL1, and XIAP. This
    contributes to the apoptosis resistance characteristic of CLL cells.
  biological_processes:
  - preferred_term: apoptotic process
    modifier: DECREASED
    term:
      id: GO:0006915
      label: apoptotic process
  downstream:
  - target: CLL Cell Accumulation
    description: Anti-apoptotic signaling enables CLL cell accumulation
- name: CLL Cell Accumulation
  description: >-
    The combination of defective apoptosis, microenvironmental support, and
    low-level proliferation leads to progressive accumulation of clonal B cells.
    CLL is primarily a disease of accumulation rather than rapid proliferation.
  cell_types:
  - preferred_term: B cell
    term:
      id: CL:0000236
      label: B cell
histopathology:
- name: Small B-Cell Clonal Expansion
  finding_term:
    preferred_term: Small Lymphocytic Lymphoma
    term:
      id: NCIT:C7540
      label: Small Lymphocytic Lymphoma
  frequency: VERY_FREQUENT
  description: Chronic lymphocytic leukemia is characterized by clonal expansion of CLL B cells.
  evidence:
  - reference: PMID:29670635
    reference_title: "Chronic Lymphocytic Leukemia B-Cell Normal Cellular Counterpart: Clues From a Functional Perspective."
    supports: SUPPORT
    snippet: "Chronic lymphocytic leukemia (CLL) is characterized by the clonal expansion of"
    explanation: Abstract describes CLL as characterized by clonal expansion of CLL B cells.

phenotypes:
- category: Hematologic
  name: Lymphoproliferative Disorder
  frequency: VERY_FREQUENT
  diagnostic: true
  description: >-
    Persistent elevation of blood lymphocyte count (at least 5000/uL clonal
    B lymphocytes) is required for CLL diagnosis. Many patients are diagnosed
    incidentally from routine blood tests.
  phenotype_term:
    preferred_term: Lymphoproliferative disorder
    term:
      id: HP:0005523
      label: Lymphoproliferative disorder
- category: Lymphatic
  name: Lymphadenopathy
  frequency: VERY_FREQUENT
  description: >-
    Enlarged lymph nodes are common, may be generalized, and can become
    massive. Lymph node involvement is incorporated in staging systems.
  phenotype_term:
    preferred_term: Lymphadenopathy
    term:
      id: HP:0002716
      label: Lymphadenopathy
  evidence:
  - reference: PMID:1139039
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "stage 1, lymphocytosis with enlarged nodes"
    explanation: The Rai staging system defines Stage I CLL by the presence of lymphadenopathy (enlarged nodes), confirming its clinical significance.
- category: Abdominal
  name: Splenomegaly
  frequency: FREQUENT
  description: >-
    Spleen enlargement from CLL infiltration. Massive splenomegaly may
    cause cytopenias from sequestration.
  phenotype_term:
    preferred_term: Splenomegaly
    term:
      id: HP:0001744
      label: Splenomegaly
  evidence:
  - reference: PMID:1139039
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "stage II, lymphocytosis with enlarged spleen or liver or both"
    explanation: The Rai staging system defines Stage II CLL by the presence of splenomegaly or hepatomegaly, confirming splenomegaly as a key clinical feature.
- category: Hematologic
  name: Anemia
  frequency: FREQUENT
  description: >-
    Anemia may result from bone marrow infiltration, autoimmune hemolytic
    anemia (AIHA), or hypersplenism.
  phenotype_term:
    preferred_term: Anemia
    term:
      id: HP:0001903
      label: Anemia
- category: Hematologic
  name: Thrombocytopenia
  frequency: FREQUENT
  description: >-
    Low platelet count from marrow infiltration, immune thrombocytopenia
    (ITP), or hypersplenism.
  phenotype_term:
    preferred_term: Thrombocytopenia
    term:
      id: HP:0001873
      label: Thrombocytopenia
- category: Infectious
  name: Recurrent Infections
  frequency: FREQUENT
  description: >-
    Increased susceptibility to bacterial and viral infections due to
    hypogammaglobulinemia and immune dysfunction. Herpes zoster and
    respiratory infections are common.
  phenotype_term:
    preferred_term: Recurrent infections
    term:
      id: HP:0002719
      label: Recurrent infections
  evidence:
  - reference: PMID:18755702
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "Patients with chronic lymphocytic leukaemia (CLL) have progressive immunodeficiency and infection is the commonest cause of death."
    explanation: This authoritative review establishes that infection due to immunodeficiency is the leading cause of death in CLL patients.
- category: Constitutional
  name: Night Sweats
  frequency: OCCASIONAL
  description: >-
    B symptoms including night sweats indicate more active disease and
    may warrant treatment initiation.
  phenotype_term:
    preferred_term: Night sweats
    term:
      id: HP:0030166
      label: Night sweats
- category: Constitutional
  name: Weight Loss
  frequency: OCCASIONAL
  description: >-
    Unintentional weight loss is a B symptom indicating active disease.
  phenotype_term:
    preferred_term: Weight loss
    term:
      id: HP:0001824
      label: Weight loss
biochemical:
- name: Flow Cytometry Immunophenotyping
  notes: >-
    CLL cells are CD5+, CD19+, CD23+, CD20 dim, surface Ig dim. The
    characteristic immunophenotype distinguishes CLL from other B-cell
    lymphomas and mantle cell lymphoma (CD5+ but CD23-).
- name: IGHV Mutation Analysis
  notes: >-
    Sequencing of immunoglobulin heavy chain variable region determines
    mutational status. Mutated IGHV (at least 2% difference from germline)
    indicates favorable prognosis.
genetic:
- name: TP53 Mutation/del(17p)
  association: High-Risk Prognostic Marker
  notes: >-
    TP53 alterations (mutation or del(17p)) occur in 5-10% at diagnosis,
    higher in relapsed disease. Confer resistance to chemoimmunotherapy
    and poor prognosis. BTK and BCL2 inhibitors remain effective.
- name: del(11q)/ATM
  association: Intermediate-Risk Marker
  notes: >-
    Deletion of 11q23 affects the ATM gene, impairing DNA damage responses.
    Associated with bulky lymphadenopathy and intermediate prognosis.
    May benefit from targeted therapies.
- name: del(13q)
  association: Favorable Prognostic Marker
  notes: >-
    Isolated del(13q14) is the most common cytogenetic abnormality in CLL
    and is associated with favorable prognosis when present alone. This
    region contains microRNA genes miR-15a and miR-16-1.
- name: IGHV
  association: Prognostic Marker
  notes: >-
    IGHV mutation status is one of the strongest prognostic markers in CLL.
    Mutated IGHV indicates post-germinal center origin with better prognosis.
    Unmutated IGHV indicates more aggressive disease course.
treatments:
- name: Ibrutinib
  description: >-
    First-in-class irreversible BTK inhibitor that disrupts BCR signaling,
    mobilizes CLL cells from tissue niches, and induces apoptosis.
    Effective in all risk groups including TP53-mutated CLL. Continuous
    therapy until progression.
  treatment_term:
    preferred_term: pharmacotherapy
    term:
      id: MAXO:0000058
      label: pharmacotherapy
    therapeutic_agent:
    - preferred_term: ibrutinib
      term:
        id: CHEBI:76612
        label: ibrutinib
  evidence:
  - reference: PMID:26639149
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "ibrutinib resulted in significantly longer progression-free survival than did chlorambucil (median, not reached vs. 18.9 months), with a risk of progression or death that was 84% lower with ibrutinib than that with chlorambucil"
    explanation: The landmark RESONATE-2 trial demonstrated ibrutinib's superiority in treatment-naive elderly CLL patients with 84% reduction in risk of progression or death.
- name: Acalabrutinib
  description: >-
    Second-generation selective BTK inhibitor with improved kinase selectivity
    and potentially fewer off-target effects than ibrutinib. Effective as
    monotherapy or combined with obinutuzumab.
  treatment_term:
    preferred_term: pharmacotherapy
    term:
      id: MAXO:0000058
      label: pharmacotherapy
    therapeutic_agent:
    - preferred_term: acalabrutinib
      term:
        id: CHEBI:167707
        label: acalabrutinib
- name: Venetoclax plus Obinutuzumab
  description: >-
    BCL2 inhibitor venetoclax combined with anti-CD20 antibody obinutuzumab
    achieves high rates of undetectable MRD and enables time-limited
    therapy (12 months). Particularly effective in treatment-naive CLL.
  treatment_term:
    preferred_term: pharmacotherapy
    term:
      id: MAXO:0000058
      label: pharmacotherapy
    therapeutic_agent:
    - preferred_term: venetoclax
      term:
        id: CHEBI:133021
        label: venetoclax
  evidence:
  - reference: PMID:30523712
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "The MURANO study demonstrated significant progression-free survival (PFS) benefit for fixed-duration venetoclax-rituximab compared with bendamustine-rituximab in relapsed/refractory chronic lymphocytic leukemia."
    explanation: The MURANO Phase III study demonstrated venetoclax-rituximab's superiority in relapsed/refractory CLL with significantly improved progression-free survival.
- name: Chemoimmunotherapy (FCR)
  description: >-
    Fludarabine, cyclophosphamide, and rituximab combination was standard
    for fit patients with mutated IGHV without del(17p)/TP53 mutation.
    Can achieve long remissions in this favorable subgroup but largely
    replaced by targeted agents.
  treatment_term:
    preferred_term: chemotherapy
    term:
      id: MAXO:0000647
      label: chemotherapy
    therapeutic_agent:
    - preferred_term: cyclophosphamide
      term:
        id: CHEBI:4027
        label: cyclophosphamide
- name: Watch and Wait
  description: >-
    Asymptomatic early-stage CLL does not require immediate treatment.
    Active surveillance with monitoring for disease progression or
    treatment indications (cytopenias, symptomatic disease, rapid
    progression) is appropriate for many patients.
  treatment_term:
    preferred_term: surveillance for malignancies
    term:
      id: MAXO:0001492
      label: surveillance for malignancies
disease_term:
  preferred_term: chronic lymphocytic leukemia
  term:
    id: MONDO:0004948
    label: B-cell chronic lymphocytic leukemia

classifications:
  icdo_morphology:
    classification_value: Leukemia
  harrisons_chapter:
  - classification_value: cancer
  - classification_value: hematologic malignancy