Acrocallosal Syndrome

Mendelian MONDO:0008708 Pathograph 24 Show in embeddings browser Ciliopathies

This entry models the autosomal recessive KIF7-related form of acrocallosal syndrome (ACLS), a rare multiple congenital anomaly disorder caused by biallelic KIF7 variants. KIF7 is the human ortholog of Drosophila Costal2 and a cilium-tip regulator of the Sonic hedgehog (SHH) pathway. It is characterized by agenesis or hypoplasia of the corpus callosum, craniofacial dysmorphism (macrocephaly, prominent forehead, hypertelorism), postaxial and/or preaxial polydactyly with duplication of the hallux, and intellectual disability. KIF7 dysfunction deregulates GLI transcription-factor output and impairs GLI3 processing, placing ACLS within the primary-cilium Hedgehog-signaling ciliopathy spectrum, with phenotypic overlap with hydrolethalus syndrome (a more severe, often lethal allelic disorder) and Joubert syndrome (shared molar tooth sign).

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1
Inheritance
5
Pathophys.
16
Phenotypes
24
Pathograph
1
Genes
3
Medical Actions
4
Differentials
13
References
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Classifications

ISDS Skeletal Nosology
polydactyly syndactyly triphalangism
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Inheritance

1
Autosomal Recessive HP:0000007
Acrocallosal syndrome is inherited in an autosomal recessive manner and is caused by biallelic (homozygous or compound heterozygous) pathogenic KIF7 variants. Truncating and splice-disrupting alleles are well established; biallelic missense substitutions have also been reported with broader, sometimes atypical phenotypes, so the allelic spectrum should not be described as exclusively loss of function.
Autosomal recessive inheritance
Show evidence (3 references)
PMID:26648833 SUPPORT Human Clinical
"Acrocallosal syndrome (ACLS) is a rare autosomal recessive disorder characterized by agenesis of the corpus callosum, facial dysmorphism, postaxial polydactyly of the hands as well as preaxial polydactyly of the feet, and developmental delay"
Establishes the autosomal recessive inheritance and the cardinal malformation tetrad of ACLS.
PMID:23125460 SUPPORT Human Clinical
"We describe the first four compound heterozygous cases"
Documents compound heterozygous KIF7 genotypes, consistent with recessive inheritance.
PMID:26174511 SUPPORT Human Clinical
"We present two children who both had two missense mutations in the Kinesin Family Member 7 (KIF7) gene."
Documents biallelic missense substitutions and prevents over-restricting the allelic spectrum to truncating loss-of-function variants.

Pathophysiology

5
KIF7 Cilium-Tip Dysfunction and Impaired Hedgehog Signal Transduction
KIF7, the human ortholog of Drosophila Costal2, is an immotile kinesin-4 protein that localizes to the tip of the primary cilium, where it organizes the cilium-tip compartment and regulates the GLI transcription factors of the Hedgehog pathway. In the absence of Hedgehog ligand KIF7 promotes processing of GLI3 into its transcriptional repressor form; upon pathway activation KIF7 and GLI accumulate at the cilium tip. Pathogenic KIF7 dysfunction deregulates GLI target genes and impairs GLI3 processing, disrupting the tightly dosed Hedgehog output required for midline brain, limb, and craniofacial patterning.
Ciliated Cell CL:0000064 Cell Ontology (CL) Relation: this pathophysiological event involves this cell type This pathophysiological event involves Ciliated Cell (CL:0000064). CL:0000064 is a cell type from the Cell Ontology.
KIF7 hgnc:30497 HUGO Gene Nomenclature Committee (hgnc) Relation: this pathophysiological event involves this gene This pathophysiological event involves KIF7 (hgnc:30497). hgnc:30497 is a gene from the HUGO Gene Nomenclature Committee.
Smoothened (Hedgehog) Signaling GO:0007224 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves dysregulated Smoothened (Hedgehog) Signaling, annotated with smoothened signaling pathway (GO:0007224). GO:0007224 is a biological process from the Gene Ontology. ↕ DYSREGULATED Regulation of Hedgehog Signaling GO:0008589 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves dysregulated Regulation of Hedgehog Signaling, annotated with regulation of smoothened signaling pathway (GO:0008589). GO:0008589 is a biological process from the Gene Ontology. ↕ DYSREGULATED
Ciliary Tip GO:0097542 Gene Ontology (GO) Relation: this pathophysiological event involves this cellular component This pathophysiological event involves Ciliary Tip (GO:0097542). GO:0097542 is a cellular component from the Gene Ontology. Primary Cilium GO:0005929 Gene Ontology (GO) Relation: this pathophysiological event involves this cellular component This pathophysiological event involves Primary Cilium, annotated with cilium (GO:0005929). GO:0005929 is a cellular component from the Gene Ontology.
Show evidence (5 references)
PMID:21552264 SUPPORT In Vitro
"KIF7, the human ortholog of Drosophila Costal2, is a key component of the Hedgehog signaling pathway"
Identifies KIF7 as a core Hedgehog pathway component (Costal2 ortholog), establishing the molecular basis of the ACLS mechanism.
PMID:21552264 SUPPORT In Vitro
"we show deregulation of most GLI transcription factor targets and impaired GLI3 processing in tissues from individuals with KIF7 mutations"
Demonstrates the downstream molecular consequence of KIF7 loss in patient tissues: deregulated GLI targets and impaired GLI3 processing.
PMID:21552264 SUPPORT In Vitro
"Our data show the role of KIF7 in human primary cilia, especially in the Hedgehog pathway through the regulation of GLI targets, and expand the clinical spectrum of ciliopathies"
Places ACLS within the primary-cilium Hedgehog-signaling ciliopathy spectrum, supporting conformance to the ciliopathy module Hedgehog node.
+ 2 more references
Dysregulated GLI3 Repressor and FGF8 Signaling
KIF7 depletion lowers GLI3-repressor signaling and overactivates SHH target genes. In the Kif7-null model, restoring GLI3-repressor activity rescues the modeled ACLS features, while increased FGF8 signaling contributes specifically to anterior corpus-callosum defects.
Smoothened Signaling Pathway GO:0007224 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves dysregulated Smoothened Signaling Pathway (GO:0007224). GO:0007224 is a biological process from the Gene Ontology. ↕ DYSREGULATED Fibroblast Growth Factor Receptor Signaling Pathway GO:0008543 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves increased Fibroblast Growth Factor Receptor Signaling Pathway (GO:0008543). GO:0008543 is a biological process from the Gene Ontology. ↑ INCREASED
Show evidence (2 references)
PMID:30445565 SUPPORT Model Organism
"decreased GLI3R signaling is fully responsible for the ACLS features in these mice, as all phenotypes are rescued by increasing GLI3R activity."
Genetic rescue in the mouse model places reduced GLI3-repressor activity downstream of Kif7 depletion.
PMID:30445565 SUPPORT Model Organism
"increased FGF8 signaling is responsible in part for CC defects associated to KIF7 depletion, as modulating FGF8 signaling rescued CC formation anteriorly in Kif7-/- mice."
Functional rescue supports increased FGF8 signaling as a partial mediator of the modeled callosal defect.
Cortical Midline Guidepost-Cell Mispatterning
In the Kif7-null model, cortical septum-boundary patterning is abnormal and guidepost cells required for callosal axons to cross the midline are redistributed. This provides an experimentally resolved route to callosal agenesis; its detailed cellular sequence remains model-organism evidence.
Corpus Callosum Development GO:0022038 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves abnormal Corpus Callosum Development (GO:0022038). GO:0022038 is a biological process from the Gene Ontology. ⚠ ABNORMAL Axon Guidance GO:0007411 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves abnormal Axon Guidance (GO:0007411). GO:0007411 is a biological process from the Gene Ontology. ⚠ ABNORMAL
Corpus Callosum UBERON:0002336 Uberon multi-species anatomy ontology (UBERON) Relation: this pathophysiological event occurs in this anatomical location This pathophysiological event occurs in Corpus Callosum (UBERON:0002336). UBERON:0002336 is an anatomical location from the Uberon multi-species anatomy ontology.
Show evidence (1 reference)
PMID:30445565 SUPPORT Model Organism
"CC agenesis in these mice is associated with specific patterning defects of the cortical septum boundary leading to altered distribution of guidepost cells required to guide the callosal axons through the midline."
Defines the cortical-midline cellular mechanism and its role in callosal axon guidance.
Cerebellar and CNS Malformation
Impaired cilium-dependent Hedgehog signaling during neurodevelopment produces the hallmark CNS malformations of ACLS: agenesis or hypoplasia of the corpus callosum and cerebellar vermis dysgenesis. In a subset of patients the cerebellar/brainstem malformation is severe enough to generate the molar tooth sign on MRI, overlapping with Joubert syndrome and reflecting the shared ciliary Hedgehog mechanism.
Neuron CL:0000540 Cell Ontology (CL) Relation: this pathophysiological event involves this cell type This pathophysiological event involves Neuron (CL:0000540). CL:0000540 is a cell type from the Cell Ontology.
Corpus Callosum Development GO:0022038 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves abnormal Corpus Callosum Development (GO:0022038). GO:0022038 is a biological process from the Gene Ontology. ⚠ ABNORMAL Cerebellum Development GO:0021549 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves abnormal Cerebellum Development (GO:0021549). GO:0021549 is a biological process from the Gene Ontology. ⚠ ABNORMAL
Corpus Callosum UBERON:0002336 Uberon multi-species anatomy ontology (UBERON) Relation: this pathophysiological event occurs in this anatomical location This pathophysiological event occurs in Corpus Callosum (UBERON:0002336). UBERON:0002336 is an anatomical location from the Uberon multi-species anatomy ontology. Cerebellar Vermis UBERON:0004720 Uberon multi-species anatomy ontology (UBERON) Relation: this pathophysiological event occurs in this anatomical location This pathophysiological event occurs in Cerebellar Vermis (UBERON:0004720). UBERON:0004720 is an anatomical location from the Uberon multi-species anatomy ontology.
Show evidence (2 references)
PMID:23125460 SUPPORT Human Clinical
"Acrocallosal syndrome (ACLS) is a rare recessive disorder characterised by corpus callosum agenesis or hypoplasia, craniofacial dysmorphism, duplication of the hallux, postaxial polydactyly, and severe mental retardation"
Establishes corpus callosum agenesis/hypoplasia as a defining CNS feature of ACLS.
PMID:23125460 SUPPORT Human Clinical
"vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly indicated the diagnosis"
Documents cerebellar vermis dysgenesis and the molar tooth sign as diagnostically informative CNS malformations in ACLS.
Limb and Craniofacial Patterning Defect
KIF7-dependent Hedgehog/GLI3 signaling is required for dosed limb and craniofacial patterning. Deregulated GLI output produces polydactyly, hallux duplication, syndactyly, cleft palate, and the characteristic macrocephalic craniofacial appearance.
limb morphogenesis GO:0035108 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves abnormal limb morphogenesis (GO:0035108). GO:0035108 is a biological process from the Gene Ontology. ⚠ ABNORMAL palate development GO:0060021 Gene Ontology (GO) Relation: this pathophysiological event involves this biological process This pathophysiological event involves abnormal palate development, annotated with roof of mouth development (GO:0060021). GO:0060021 is a biological process from the Gene Ontology. ⚠ ABNORMAL
Show evidence (2 references)
PMID:21552264 SUPPORT Human Clinical
"overlapping features that include polydactyly, brain abnormalities and cleft palate"
Original KIF7 report supports limb and craniofacial malformations as part of the KIF7 ciliopathy spectrum.
PMID:26349186 SUPPORT Human Clinical
"characterized by postaxial and/or preaxial polydactyly, cutaneous syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of the corpus callosum, and intellectual disability"
Review evidence connects KIF7-associated ACLS to polydactyly, syndactyly, macrocephaly, and hypertelorism.

Pathograph

Use the checkboxes to hide or show graph categories. Hover nodes for evidence and cross-linked metadata.
Pathograph: causal mechanism network for Acrocallosal Syndrome Interactive directed graph showing how pathophysiology mechanisms, phenotypes, genetic factors and variants, experimental models, environmental triggers, and treatments relate through causal and linked edges.

Phenotypes

16
Eye 1
Hypertelorism VERY_FREQUENT HP:0000316 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Hypertelorism (HP:0000316). HP:0000316 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:23125460 SUPPORT Human Clinical
"Facial dysmorphism with hypertelorism and prominent forehead in all the cases"
Hypertelorism was present in all cases of this ACLS series, supporting VERY_FREQUENT classification.
Head and Neck 3
Macrocephaly HP:0000256 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Macrocephaly (HP:0000256). HP:0000256 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26349186 SUPPORT Human Clinical
"characterized by postaxial and/or preaxial polydactyly, cutaneous syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of the corpus callosum, and intellectual disability"
Macrocephaly is enumerated among the characteristic features of ACLS.
Prominent Forehead VERY_FREQUENT HP:0011220 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Prominent forehead (HP:0011220). HP:0011220 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:23125460 SUPPORT Human Clinical
"Facial dysmorphism with hypertelorism and prominent forehead in all the cases"
Prominent forehead was present in all cases of this ACLS series, supporting VERY_FREQUENT classification.
Cleft Palate HP:0000175 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Cleft palate (HP:0000175). HP:0000175 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:21552264 SUPPORT Human Clinical
"polydactyly, brain abnormalities and cleft palate"
Cleft palate is listed among the overlapping malformation features of hydrolethalus and acrocallosal syndromes in the original KIF7 discovery paper.
Limbs 2
Postaxial Polydactyly Postaxial hand polydactyly HP:0001162 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Postaxial hand polydactyly (HP:0001162). HP:0001162 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26349186 SUPPORT Human Clinical
"Acrocallosal syndrome is a multiple congenital anomaly disorder characterized by postaxial and/or preaxial polydactyly"
Postaxial polydactyly is a cardinal limb malformation of ACLS.
Cutaneous Syndactyly HP:0012725 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Cutaneous syndactyly (HP:0012725). HP:0012725 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26349186 SUPPORT Human Clinical
"characterized by postaxial and/or preaxial polydactyly, cutaneous syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of the corpus callosum, and intellectual disability"
Cutaneous syndactyly is enumerated among the characteristic limb features of ACLS in this KIF7 syndromology review.
Musculoskeletal 1
Hypotonia HP:0001252 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Hypotonia (HP:0001252). HP:0001252 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:22696705 SUPPORT Human Clinical
"Except for hypotonia, rest of the central nervous system examination was normal."
Documents hypotonia in a clinically diagnosed adult case.
Nervous System 6
Agenesis of Corpus Callosum HP:0001274 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Agenesis of corpus callosum (HP:0001274). HP:0001274 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26648833 SUPPORT Human Clinical
"Acrocallosal syndrome (ACLS) is a rare autosomal recessive disorder characterized by agenesis of the corpus callosum"
Corpus callosum agenesis is the eponymous, defining CNS malformation of ACLS.
Hypoplasia of Corpus Callosum Hypoplasia of the corpus callosum HP:0002079 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Hypoplasia of the corpus callosum (HP:0002079). HP:0002079 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:23125460 SUPPORT Human Clinical
"Acrocallosal syndrome (ACLS) is a rare recessive disorder characterised by corpus callosum agenesis or hypoplasia"
Explicitly documents hypoplasia as part of the callosal phenotype rather than collapsing all cases into complete agenesis.
Intellectual Disability HP:0001249 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Intellectual disability (HP:0001249). HP:0001249 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26349186 SUPPORT Human Clinical
"absence or hypoplasia of the corpus callosum, and intellectual disability"
Intellectual disability is a defining neurodevelopmental feature of ACLS.
Seizure FREQUENT HP:0001250 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Seizure (HP:0001250). HP:0001250 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:29321670 SUPPORT Human Clinical
"Seizure7/18 (39%)2/19 (11%)"
The KIF7-positive ACLS/Joubert spectrum had seizures in 7 of 18 individuals with available data, supporting the FREQUENT band.
Cerebellar Vermis Hypoplasia HP:0001320 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Cerebellar vermis hypoplasia (HP:0001320). HP:0001320 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:23125460 SUPPORT Human Clinical
"vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly indicated the diagnosis"
Cerebellar vermis dysgenesis is a CNS malformation supporting the diagnosis of ACLS.
Molar Tooth Sign Molar tooth sign on MRI HP:0002419 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Molar tooth sign on MRI (HP:0002419). HP:0002419 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26349186 SUPPORT Human Clinical
"They present with features like molar tooth sign and hyperventilation that are not very typical in ACLS, but do occur in other ciliopathies"
The molar tooth sign is described as "not very typical" in ACLS, supporting its presence in a variable subset without assigning a frequency from qualitative wording.
Other 3
Preaxial Polydactyly Preaxial foot polydactyly HP:0001841 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Preaxial foot polydactyly (HP:0001841). HP:0001841 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:26648833 SUPPORT Human Clinical
"postaxial polydactyly of the hands as well as preaxial polydactyly of the feet"
Preaxial polydactyly of the feet is a recognized cardinal limb feature of ACLS.
Duplication of the Hallux Duplication of phalanx of hallux HP:0010066 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Duplication of the hallux, annotated with Duplication of phalanx of hallux (HP:0010066). HP:0010066 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:23125460 SUPPORT Human Clinical
"corpus callosum agenesis or hypoplasia, craniofacial dysmorphism, duplication of the hallux, postaxial polydactyly"
Hallux duplication is listed among the defining clinical features of ACLS.
Dandy-Walker Malformation HP:0001305 Human Phenotype Ontology (HP) Relation: this clinical feature is this phenotype This clinical feature is Dandy-Walker malformation (HP:0001305). HP:0001305 is a phenotype from the Human Phenotype Ontology.
Show evidence (1 reference)
PMID:29910499 SUPPORT Human Clinical
"showed partial callosal agenesis, pachygyria, ventriculomegaly, agenesis of cerebellar vermis and Dandy–Walker malformation."
A KIF7-confirmed infant had Dandy-Walker malformation on MRI, supporting inclusion without assigning a population frequency.
🧬

Genetic Associations

1
Biallelic KIF7 Variants (Causative)
Gene: KIF7 hgnc:30497 HUGO Gene Nomenclature Committee (hgnc) Relation: this disease-associated gene is this gene This disease-associated gene is KIF7 (hgnc:30497). hgnc:30497 is a gene from the HUGO Gene Nomenclature Committee. relationship_type: CAUSATIVE variant_origin: GERMLINE
Show evidence (4 references)
PMID:21552264 SUPPORT Human Clinical
"we report mutations in KIF7 in individuals with hydrolethalus and acrocallosal syndromes, two multiple malformation disorders with overlapping features that include polydactyly, brain abnormalities and cleft palate"
Original report identifying KIF7 as the causative gene for both ACLS and the allelic hydrolethalus syndrome.
PMID:23125460 SUPPORT Human Clinical
"Seven mutations were identified at the KIF7 locus in these five cases, six of which are novel"
Mutation series confirming KIF7 as the recurrent disease locus and expanding the allelic spectrum of ACLS.
PMID:26648833 SUPPORT Human Clinical
"Mutations in the KIF7 gene, encoding a molecule within the Sonic hedgehog (SHH) pathway, have been identified as causative for ACLS but also for the fatal hydrolethalus syndrome and some cases of Joubert syndrome"
Confirms KIF7 causation of ACLS and the allelic relationship with hydrolethalus and Joubert syndromes.
+ 1 more reference
💊

Medical Actions

3
Supportive and Multidisciplinary Care
Category: Therapeutic Action: supportive careNCI Thesaurus (NCIT) Relation: this treatment is this clinical intervention This treatment is supportive care (NCIT:C15747). NCIT:C15747 is a clinical intervention from the NCI Thesaurus. Ontology label: Supportive Care NCIT:C15747
Management is symptom-directed and multidisciplinary. Early physical, occupational, developmental, and communication support addresses hypotonia and developmental disability; seizures are treated with standard antiseizure therapy when present. Available evidence is case-report and clinical-review level rather than ACLS-specific comparative trials.
Target Phenotypes: Hypotonia HP:0001252 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Hypotonia (HP:0001252). HP:0001252 is a phenotype from the Human Phenotype Ontology. Intellectual disability HP:0001249 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Intellectual disability (HP:0001249). HP:0001249 is a phenotype from the Human Phenotype Ontology. Seizure HP:0001250 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Seizure (HP:0001250). HP:0001250 is a phenotype from the Human Phenotype Ontology.
Show evidence (3 references)
PMID:22696705 SUPPORT Human Clinical
"Treatment includes physical therapy in infancy which may assist in the development of motor skills and muscle tone."
Supports early physical therapy for hypotonia and motor delay.
PMID:22696705 SUPPORT Human Clinical
"Antiepileptic therapy for seizure disorder and antihypertensives for renoparenchymal hypertension are to be given."
Supports symptom-directed antiseizure treatment when seizures occur.
PMID:29910499 SUPPORT Human Clinical
"At present, the child is referred to a neurodevelopment specialist, for neurodevelopment assessment and occupational therapist for infant stimulation program."
A genetically confirmed case documents specialist developmental follow-up and occupational-therapy referral.
Surgical Correction of Polydactyly and Malformations
Category: Therapeutic Action: surgical procedureNCI Thesaurus (NCIT) Relation: this treatment is this clinical intervention This treatment is surgical procedure (NCIT:C15329). NCIT:C15329 is a clinical intervention from the NCI Thesaurus. Ontology label: Surgical Procedure NCIT:C15329
Individually indicated surgery may remove functionally limiting extra digits, release syndactyly, or repair orofacial and other structural malformations. These procedures treat local manifestations rather than the underlying ciliary signaling defect.
Target Phenotypes: Postaxial hand polydactyly HP:0001162 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Postaxial hand polydactyly (HP:0001162). HP:0001162 is a phenotype from the Human Phenotype Ontology. Preaxial foot polydactyly HP:0001841 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Preaxial foot polydactyly (HP:0001841). HP:0001841 is a phenotype from the Human Phenotype Ontology. Cutaneous syndactyly HP:0012725 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Cutaneous syndactyly (HP:0012725). HP:0012725 is a phenotype from the Human Phenotype Ontology. Cleft palate HP:0000175 Human Phenotype Ontology (HP) Relation: this treatment targets this phenotype This treatment targets Cleft palate (HP:0000175). HP:0000175 is a phenotype from the Human Phenotype Ontology.
Show evidence (2 references)
PMID:22696705 SUPPORT Human Clinical
"Surgery to remove extra fingers and toes and release fused fingers may improve movement and grasp, may assist in walking and the comfort of footwear."
Supports symptom-directed surgery for polydactyly and syndactyly while making no disease-modifying claim.
PMID:29910499 SUPPORT Human Clinical
"She underwent Nissen fundoplication at 4 months of age, cleft lip repair at 6 months age and cleft palate repair at 18 months of age using the same technique."
A KIF7-confirmed case documents actual surgical repair of associated structural manifestations, including cleft palate.
Genetic Counseling
Category: Counseling / Informational Action: Genetic CounselingNCI Thesaurus (NCIT) Relation: this treatment is this clinical intervention This treatment is Genetic Counseling (NCIT:C15240). NCIT:C15240 is a clinical intervention from the NCI Thesaurus. NCIT:C15240
When biallelic KIF7 disease is confirmed, parents should be counseled about autosomal recessive inheritance and a 25% recurrence risk in each pregnancy. Prenatal ultrasound can detect polydactyly and major CNS malformations but cannot exclude disease because presentation is variable. This recurrence statement does not apply to a rare de novo GLI3 phenocopy.
Show evidence (2 references)
PMID:24949054 SUPPORT Human Clinical
"As the mode of inheritance of ACLS is autosomal recessive, the risk of recurrence is 25%. Genetic counselling is of prime importance"
Directly supports recurrence-risk counseling for the autosomal recessive KIF7 form.
PMID:24949054 SUPPORT Human Clinical
"Polydactyly, and central nervous system malformations can be detected by ultrasonography in the second trimester, but due to variability of presentation, prenatal diagnosis may not always be possible."
Supports balanced counseling about prenatal ultrasound capabilities and limits.
🔬

Diagnosis

2
Clinical and Molecular Diagnosis
Acrocallosal syndrome is suspected from the combination of corpus callosum agenesis/hypoplasia, craniofacial dysmorphism (macrocephaly, prominent forehead, hypertelorism), and polydactyly, and is confirmed by identification of biallelic KIF7 pathogenic variants. Because expressivity is variable, KIF7 testing is warranted in patients with suggestive craniofacial features even when polydactyly or callosal anomalies are absent. If targeted KIF7 testing is negative, a broader ciliopathy/ polydactyly panel or exome analysis can evaluate phenocopies involving CPLANE1/C5orf42, GLI3, SHH, and other developmental-pathway genes.
molecular genetic testing NCIT:C19770 NCI Thesaurus (NCIT)
Show evidence (3 references)
PMID:23125460 SUPPORT Human Clinical
"KIF7 should be tested in less typical patients in whom craniofacial features are suggestive of ACLS"
Defines the molecular diagnostic recommendation for ACLS, including in atypical presentations.
PMID:29321670 SUPPORT Human Clinical
"Genetic analysis in our cohort of patients with craniofacial appearance fitting into the ACLS clinical spectrum and variable additional ciliopathy features revealed (likely) pathogenic variants not only in KIF7, but also secondly in C5orf42"
Demonstrates locus heterogeneity in ACLS-like presentations and supports broader sequencing when KIF7 testing is unrevealing.
PMID:29321670 SUPPORT Human Clinical
"In addition, in one patient we found a heterozygous de novo frameshift variant in SHH, the most common cause of non-chromosomal holoprosencephaly (HPE)"
Supports inclusion of SHH-related phenocopies in a broader negative-KIF7 diagnostic workup.
Prenatal Ultrasound Assessment
In an at-risk pregnancy, second-trimester ultrasound can identify polydactyly and major central-nervous-system malformations, but variable expressivity means a normal scan does not exclude KIF7-related disease.
clinical imaging procedure NCIT:C17369 NCI Thesaurus (NCIT)
Show evidence (1 reference)
PMID:24949054 SUPPORT Human Clinical
"Polydactyly, and central nervous system malformations can be detected by ultrasonography in the second trimester, but due to variability of presentation, prenatal diagnosis may not always be possible."
Supports both the detectable prenatal features and the limitation of ultrasound caused by variable presentation.
🩻

Imaging Findings

3
Corpus Callosum Agenesis or Hypoplasia on MRI
Brain MRI demonstrates complete agenesis or partial hypoplasia of the corpus callosum. This is a cardinal diagnostic component of the syndrome, although molecular confirmation is required for KIF7-specific diagnosis.
Mri Diagnostic
Abnormal corpus callosum morphology HP:0001273 Human Phenotype Ontology (HP) Corpus Callosum UBERON:0002336 Uberon multi-species anatomy ontology (UBERON) Abnormal corpus callosum morphology HP:0001273 Human Phenotype Ontology (HP)
Show evidence (3 references)
PMID:23125460 SUPPORT Human Clinical
"corpus callosum agenesis or hypoplasia, craniofacial dysmorphism, duplication of the hallux, postaxial polydactyly"
Establishes callosal agenesis or hypoplasia as a defining structural neuroimaging finding.
PMID:29910499 SUPPORT Human Clinical
"Magnetic resonance imaging (MRI) of the brain"
Establishes MRI as the modality used for the structural brain findings in this KIF7-confirmed case.
PMID:29910499 SUPPORT Human Clinical
"showed partial callosal agenesis, pachygyria, ventriculomegaly, agenesis of cerebellar vermis and Dandy–Walker malformation."
The same MRI sentence documents partial callosal agenesis in a KIF7-confirmed case.
Molar Tooth Sign on MRI
Vermis dysgenesis with associated brainstem configuration can create the molar tooth sign. It occurs in a variable subset and signals overlap with the KIF7-related Joubert spectrum rather than defining every ACLS case.
Mri
Molar tooth sign on MRI HP:0002419 Human Phenotype Ontology (HP) Brainstem UBERON:0002298 Uberon multi-species anatomy ontology (UBERON) Molar tooth sign on MRI HP:0002419 Human Phenotype Ontology (HP)
Show evidence (1 reference)
PMID:23125460 SUPPORT Human Clinical
"vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly indicated the diagnosis"
Documents the molar tooth configuration on brain imaging.
Olfactory Bulb and Tract Abnormalities on Neuroimaging
Olfactory-bulb and olfactory-tract abnormalities were reported on neuroimaging in one child with a KIF7-confirmed acrocallosal phenotype. The available abstract does not isolate the modality for this finding, so none is asserted here. This is an emerging single-case extension, not a defining or frequency-estimated imaging feature.
Single KIF7-confirmed case report
Olfactory Bulb UBERON:0002264 Uberon multi-species anatomy ontology (UBERON)
Show evidence (1 reference)
PMID:31399769 SUPPORT Human Clinical
"We describe association of olfactory bulb and olfactory tract abnormalities in a child with acrocallosal syndrome caused by kinesin family membrane 7 (KIF7) mutation in sonic hedgehog pathway."
Molecularly confirmed single-case evidence supports this explicitly qualified imaging extension.
📈

Progression

2
Congenital onset
Age: Antenatal to neonatal
The structural brain, craniofacial, and limb anomalies arise prenatally and are present at birth, although the full neurodevelopmental phenotype becomes apparent during follow-up.
Show evidence (1 reference)
PMID:24949054 SUPPORT Human Clinical
"An Indian neonate presented on day 1 of life (youngest in the literature to be reported) with combination of abnormalities consistent with the acrocallosal syndrome and some additional findings."
A neonatal case directly establishes congenital presentation.
Neurodevelopmental course
Age: Childhood to adulthood
Developmental impairment is usually substantial but varies across the KIF7-associated spectrum. Long-term observations include persistent craniofacial dysmorphism and severe intellectual disability in many KIF7-positive individuals, while milder adult cognitive outcomes have been reported for some alleles.
Show evidence (2 references)
PMID:29321670 SUPPORT Human Clinical
"However, long-term follow-up revealed that patients with pathogenic KIF7 variants had more severe and persistent facial dysmorphism and in general more severe ID, while in patients with (likely) pathogenic C5orf42 variants, facial dysmorphism became less prominent over time and they could catch..."
Long-term comparative follow-up documents persistence and variability in the KIF7-associated neurodevelopmental course.
PMID:29321670 SUPPORT Human Clinical
"Importantly, patient 3 with homozygosity of the KIF7 splice site pathogenic variant at position −3 could also catch up with only mild cognitive deficits in adulthood"
Directly supports a milder adult cognitive outcome for one KIF7 splice variant while avoiding a broad genotype-phenotype rule.
📊

Prevalence

1
Worldwide
Cases In Literature Ultra Rare
A historical case report and literature summary identified only 37 previously described cases worldwide. This is a historical literature-case count for the clinically defined phenotype across genetic etiologies, not a KIF7-specific or denominator-based population estimate, and the current total is expected to be higher.
Show evidence (1 reference)
PMID:22696705 SUPPORT Human Clinical
"Since Schinzel’s original description, only 37 cases of acrocallosal syndrome have been described worldwide."
Supports a historical worldwide cases-in-literature classification while avoiding conversion of the count into an epidemiologic rate.
🔀

Differential Diagnoses

4

Conditions with similar clinical presentations that must be differentiated from Acrocallosal Syndrome:

Overlapping Features Joubert syndrome shares the molar tooth sign and is caused in a subset of cases by KIF7 mutations; distinguished by the predominance of the cerebellar/brainstem malformation and characteristic abnormal eye movements and breathing dysregulation.
Distinguishing Features
  • A predominant molar-tooth imaging phenotype with oculomotor apraxia or breathing dysregulation favors a Joubert-spectrum diagnosis over classic ACLS.
Show evidence (3 references)
PMID:26648833 SUPPORT Human Clinical
"have been identified as causative for ACLS but also for the fatal hydrolethalus syndrome and some cases of Joubert syndrome"
Documents the KIF7 allelic overlap between ACLS and Joubert syndrome, relevant to differential diagnosis.
PMID:26349186 SUPPORT Human Clinical
"They present with features like molar tooth sign and hyperventilation that are not very typical in ACLS, but do occur in other ciliopathies"
Supports the molar-tooth and breathing-regulation distinction from classic ACLS.
PMID:29321670 SUPPORT Human Clinical
"none of the KIF7 patients but all the C5orf42 patients in our cohort presented with ocular motor apraxia, which is commonly associated with the MTS in JBTS patients"
Supports oculomotor apraxia as a discriminator in ACLS-like versus Joubert-spectrum presentations while preserving gene-specific context.
{ }

Source YAML

click to show
name: Acrocallosal Syndrome
synonyms:
- Acrocallosal syndrome, KIF7-related
creation_date: "2026-06-13T20:27:42Z"
category: Mendelian
description: >-
  This entry models the autosomal recessive KIF7-related form of acrocallosal
  syndrome (ACLS), a rare multiple congenital anomaly disorder caused by
  biallelic KIF7 variants. KIF7 is the human ortholog of Drosophila Costal2 and
  a cilium-tip regulator of the Sonic hedgehog (SHH) pathway. It is
  characterized by agenesis or hypoplasia of the
  corpus callosum, craniofacial dysmorphism (macrocephaly, prominent forehead,
  hypertelorism), postaxial and/or preaxial polydactyly with duplication of the
  hallux, and intellectual disability. KIF7 dysfunction deregulates GLI
  transcription-factor output and impairs GLI3 processing, placing ACLS within
  the primary-cilium Hedgehog-signaling ciliopathy spectrum, with phenotypic
  overlap with hydrolethalus syndrome (a more severe, often lethal allelic
  disorder) and Joubert syndrome (shared molar tooth sign).
disease_term:
  preferred_term: acrocallosal syndrome
  term:
    id: MONDO:0008708
    label: acrocallosal syndrome
parents:
- Ciliopathies
inheritance:
- name: Autosomal Recessive
  inheritance_term:
    preferred_term: Autosomal recessive inheritance
    term:
      id: HP:0000007
      label: Autosomal recessive inheritance
  description: >-
    Acrocallosal syndrome is inherited in an autosomal recessive manner and is
    caused by biallelic (homozygous or compound heterozygous) pathogenic KIF7
    variants. Truncating and splice-disrupting alleles are well established;
    biallelic missense substitutions have also been reported with broader,
    sometimes atypical phenotypes, so the allelic spectrum should not be
    described as exclusively loss of function.
  evidence:
  - reference: PMID:26648833
    reference_title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Acrocallosal syndrome (ACLS) is a rare autosomal recessive disorder
      characterized by agenesis of the corpus callosum, facial dysmorphism,
      postaxial polydactyly of the hands as well as preaxial polydactyly of the
      feet, and developmental delay
    explanation: >-
      Establishes the autosomal recessive inheritance and the cardinal
      malformation tetrad of ACLS.
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      We describe the first four compound heterozygous cases
    explanation: >-
      Documents compound heterozygous KIF7 genotypes, consistent with recessive
      inheritance.
  - reference: PMID:26174511
    reference_title: "Novel KIF7 missense substitutions in two patients presenting with multiple malformations and features of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      We present two children who both had two missense mutations in the Kinesin
      Family Member 7 (KIF7) gene.
    explanation: >-
      Documents biallelic missense substitutions and prevents over-restricting
      the allelic spectrum to truncating loss-of-function variants.
classifications:
  isds_skeletal_category:
  - classification_value: polydactyly_syndactyly_triphalangism
    notes: >-
      ISDS Nosology and Classification of Genetic Skeletal Disorders, 2019 revision
      (Mortier et al., PMID:31633310), Table 1 group 41
      "Polydactyly-Syndactyly-Triphalangism group"; listed as "Acrocallosal syndrome".
prevalence:
- population: Worldwide
  measure_type: CASES_IN_LITERATURE
  prevalence_class: ULTRA_RARE
  notes: >-
    A historical case report and literature summary identified only 37
    previously described cases worldwide. This is a historical literature-case
    count for the clinically defined phenotype across genetic etiologies, not a
    KIF7-specific or denominator-based population estimate, and the current
    total is expected to be higher.
  evidence:
  - reference: PMID:22696705
    reference_title: "Acrocallosal syndrome in a young hypertensive male."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Since Schinzel’s original description, only 37 cases of acrocallosal
      syndrome have been described worldwide.
    explanation: >-
      Supports a historical worldwide cases-in-literature classification while
      avoiding conversion of the count into an epidemiologic rate.
progression:
- phase: Congenital onset
  age_range: Antenatal to neonatal
  notes: >-
    The structural brain, craniofacial, and limb anomalies arise prenatally and
    are present at birth, although the full neurodevelopmental phenotype becomes
    apparent during follow-up.
  evidence:
  - reference: PMID:24949054
    reference_title: "The acrocallosal syndrome in a neonate with further widening of phenotypic expression."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      An Indian neonate presented on day 1 of life (youngest in the literature
      to be reported) with combination of abnormalities consistent with the
      acrocallosal syndrome and some additional findings.
    explanation: A neonatal case directly establishes congenital presentation.
- phase: Neurodevelopmental course
  age_range: Childhood to adulthood
  notes: >-
    Developmental impairment is usually substantial but varies across the
    KIF7-associated spectrum. Long-term observations include persistent
    craniofacial dysmorphism and severe intellectual disability in many
    KIF7-positive individuals, while milder adult cognitive outcomes have been
    reported for some alleles.
  evidence:
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      However, long-term follow-up revealed that patients with pathogenic KIF7
      variants had more severe and persistent facial dysmorphism and in general
      more severe ID, while in patients with (likely) pathogenic C5orf42
      variants, facial dysmorphism became less prominent over time and they
      could catch up and reach a level of mild to moderate ID despite initial
      severe DD.
    explanation: >-
      Long-term comparative follow-up documents persistence and variability in
      the KIF7-associated neurodevelopmental course.
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Importantly, patient 3 with homozygosity of the KIF7 splice site
      pathogenic variant at position −3 could also catch up with only mild
      cognitive deficits in adulthood
    explanation: >-
      Directly supports a milder adult cognitive outcome for one KIF7 splice
      variant while avoiding a broad genotype-phenotype rule.
pathophysiology:
- name: KIF7 Cilium-Tip Dysfunction and Impaired Hedgehog Signal Transduction
  conforms_to: "ciliopathy_dysfunction#Impaired Hedgehog Signal Transduction"
  description: >-
    KIF7, the human ortholog of Drosophila Costal2, is an immotile kinesin-4
    protein that localizes to the tip of the primary cilium, where it organizes
    the cilium-tip compartment and regulates the GLI transcription factors of
    the Hedgehog pathway. In the absence of Hedgehog ligand KIF7 promotes
    processing of GLI3 into its transcriptional repressor form; upon pathway
    activation KIF7 and GLI accumulate at the cilium tip. Pathogenic KIF7
    dysfunction deregulates GLI target genes and impairs GLI3 processing,
    disrupting the tightly dosed Hedgehog output required for midline brain,
    limb, and craniofacial patterning.
  genes:
  - preferred_term: KIF7
    term:
      id: hgnc:30497
      label: KIF7
  biological_processes:
  - preferred_term: Smoothened (Hedgehog) Signaling
    term:
      id: GO:0007224
      label: smoothened signaling pathway
    modifier: DYSREGULATED
  - preferred_term: Regulation of Hedgehog Signaling
    term:
      id: GO:0008589
      label: regulation of smoothened signaling pathway
    modifier: DYSREGULATED
  cellular_components:
  - preferred_term: Ciliary Tip
    term:
      id: GO:0097542
      label: ciliary tip
  - preferred_term: Primary Cilium
    term:
      id: GO:0005929
      label: cilium
  cell_types:
  - preferred_term: Ciliated Cell
    term:
      id: CL:0000064
      label: ciliated cell
  downstream:
  - target: Dysregulated GLI3 Repressor and FGF8 Signaling
    causal_link_type: DIRECT
    description: >-
      KIF7 depletion impairs GLI3 processing, reduces GLI3 repressor activity,
      overactivates SHH target genes, and increases FGF8 signaling.
    evidence:
    - reference: PMID:30445565
      reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
      supports: SUPPORT
      evidence_source: MODEL_ORGANISM
      snippet: >-
        In both humans and mice, KIF7 depletion leads to abnormal GLI3
        processing and over-activation of SHH target genes.
      explanation: >-
        Directly links KIF7 depletion to the altered GLI output represented by
        the downstream signaling node.
    - reference: PMID:30445565
      reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
      supports: SUPPORT
      evidence_source: MODEL_ORGANISM
      snippet: >-
        increased FGF8 signaling is responsible in part for CC defects
        associated to KIF7 depletion
      explanation: >-
        Completes the edge-level evidence for the FGF8 component of the
        downstream signaling node.
  evidence:
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      KIF7, the human ortholog of Drosophila Costal2, is a key component of the
      Hedgehog signaling pathway
    explanation: >-
      Identifies KIF7 as a core Hedgehog pathway component (Costal2 ortholog),
      establishing the molecular basis of the ACLS mechanism.
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      we show deregulation of most GLI transcription factor targets and impaired
      GLI3 processing in tissues from individuals with KIF7 mutations
    explanation: >-
      Demonstrates the downstream molecular consequence of KIF7 loss in patient
      tissues: deregulated GLI targets and impaired GLI3 processing.
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      Our data show the role of KIF7 in human primary cilia, especially in the
      Hedgehog pathway through the regulation of GLI targets, and expand the
      clinical spectrum of ciliopathies
    explanation: >-
      Places ACLS within the primary-cilium Hedgehog-signaling ciliopathy
      spectrum, supporting conformance to the ciliopathy module Hedgehog node.
  - reference: PMID:34705483
    reference_title: "Hedgehog-induced ciliary trafficking of kinesin-4 motor KIF7 requires intraflagellar transport but not KIF7's microtubule binding."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      In vertebrates, Hedgehog signaling requires the primary cilium, and KIF7
      and Gli transcription factors accumulate at the cilium tip in response to
      Hedgehog activation
    explanation: >-
      Cell-based evidence that KIF7 is a cilium-tip Hedgehog regulator whose
      ciliary accumulation is coupled to pathway activation.
  - reference: PMID:34705483
    reference_title: "Hedgehog-induced ciliary trafficking of kinesin-4 motor KIF7 requires intraflagellar transport but not KIF7's microtubule binding."
    supports: SUPPORT
    evidence_source: IN_VITRO
    snippet: >-
      we show that the immotile behavior of KIF7 is required to prevent ciliary
      localization of Gli transcription factors in the absence of Hedgehog
      signaling
    explanation: >-
      Mechanistic detail that KIF7 restrains GLI in the unstimulated state,
      explaining how loss of function deregulates GLI output.
- name: Dysregulated GLI3 Repressor and FGF8 Signaling
  description: >-
    KIF7 depletion lowers GLI3-repressor signaling and overactivates SHH target
    genes. In the Kif7-null model, restoring GLI3-repressor activity rescues the
    modeled ACLS features, while increased FGF8 signaling contributes
    specifically to anterior corpus-callosum defects.
  biological_processes:
  - preferred_term: Smoothened Signaling Pathway
    term:
      id: GO:0007224
      label: smoothened signaling pathway
    modifier: DYSREGULATED
  - preferred_term: Fibroblast Growth Factor Receptor Signaling Pathway
    term:
      id: GO:0008543
      label: fibroblast growth factor receptor signaling pathway
    modifier: INCREASED
  evidence:
  - reference: PMID:30445565
    reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
    supports: SUPPORT
    evidence_source: MODEL_ORGANISM
    snippet: >-
      decreased GLI3R signaling is fully responsible for the ACLS features in
      these mice, as all phenotypes are rescued by increasing GLI3R activity.
    explanation: >-
      Genetic rescue in the mouse model places reduced GLI3-repressor activity
      downstream of Kif7 depletion.
  - reference: PMID:30445565
    reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
    supports: SUPPORT
    evidence_source: MODEL_ORGANISM
    snippet: >-
      increased FGF8 signaling is responsible in part for CC defects associated
      to KIF7 depletion, as modulating FGF8 signaling rescued CC formation
      anteriorly in Kif7-/- mice.
    explanation: >-
      Functional rescue supports increased FGF8 signaling as a partial mediator
      of the modeled callosal defect.
  downstream:
  - target: Cortical Midline Guidepost-Cell Mispatterning
    causal_link_type: INDIRECT_KNOWN_INTERMEDIATES
    intermediate_mechanisms:
    - cortical septum-boundary patterning defects
    description: >-
      Altered GLI3-repressor and FGF8 signaling disrupts cortical septum-boundary
      patterning and the distribution of guidepost cells at the callosal
      midline.
    evidence:
    - reference: PMID:30445565
      reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
      supports: SUPPORT
      evidence_source: MODEL_ORGANISM
      snippet: >-
        CC agenesis in these mice is associated with specific patterning defects
        of the cortical septum boundary leading to altered distribution of
        guidepost cells required to guide the callosal axons through the
        midline.
      explanation: >-
        The Kif7-null model directly connects signaling disruption with the
        cortical-boundary and guidepost-cell defect.
  - target: Cerebellar and CNS Malformation
    causal_link_type: INDIRECT_UNKNOWN_INTERMEDIATES
    description: >-
      Developmentally mis-dosed GLI signaling is consistent with the
      cerebellar and broader CNS malformation spectrum, but the intervening
      tissue-specific sequence has not been resolved.
    evidence:
    - reference: PMID:21552264
      reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
      supports: SUPPORT
      evidence_source: IN_VITRO
      snippet: >-
        we show deregulation of most GLI transcription factor targets and
        impaired GLI3 processing in tissues from individuals with KIF7
        mutations
      explanation: >-
        Patient-tissue evidence supports the signaling lesion, but does not by
        itself resolve the downstream CNS-developmental sequence.
    - reference: PMID:21552264
      reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        overlapping features that include polydactyly, brain abnormalities and
        cleft palate
      explanation: >-
        Human genetic evidence supports the target brain-malformation spectrum;
        the edge remains explicitly indirect with unknown intermediates.
  - target: Limb and Craniofacial Patterning Defect
    causal_link_type: INDIRECT_KNOWN_INTERMEDIATES
    intermediate_mechanisms:
    - reduced GLI3-repressor-dependent digit patterning
    - abnormal embryonic craniofacial patterning
    description: >-
      Altered GLI3 output disrupts anterior-posterior digit specification and
      midline craniofacial development.
    evidence:
    - reference: PMID:30445565
      reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
      supports: SUPPORT
      evidence_source: MODEL_ORGANISM
      snippet: >-
        decreased GLI3R signaling is fully responsible for the ACLS features in
        these mice, as all phenotypes are rescued by increasing GLI3R activity.
      explanation: >-
        Genetic restoration of GLI3-repressor activity rescued the modeled ACLS
        features, including the polydactyly phenotype reported in this model.
- name: Cortical Midline Guidepost-Cell Mispatterning
  description: >-
    In the Kif7-null model, cortical septum-boundary patterning is abnormal and
    guidepost cells required for callosal axons to cross the midline are
    redistributed. This provides an experimentally resolved route to callosal
    agenesis; its detailed cellular sequence remains model-organism evidence.
  biological_processes:
  - preferred_term: Corpus Callosum Development
    term:
      id: GO:0022038
      label: corpus callosum development
    modifier: ABNORMAL
  - preferred_term: Axon Guidance
    term:
      id: GO:0007411
      label: axon guidance
    modifier: ABNORMAL
  locations:
  - preferred_term: Corpus Callosum
    term:
      id: UBERON:0002336
      label: corpus callosum
  evidence:
  - reference: PMID:30445565
    reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
    supports: SUPPORT
    evidence_source: MODEL_ORGANISM
    snippet: >-
      CC agenesis in these mice is associated with specific patterning defects
      of the cortical septum boundary leading to altered distribution of
      guidepost cells required to guide the callosal axons through the midline.
    explanation: >-
      Defines the cortical-midline cellular mechanism and its role in callosal
      axon guidance.
  downstream:
  - target: Agenesis of Corpus Callosum
    causal_link_type: INDIRECT_KNOWN_INTERMEDIATES
    intermediate_mechanisms:
    - failed guidance of callosal axons across the cortical midline
    description: >-
      Mispatterned midline guidepost cells are associated with failed callosal
      axon guidance and agenesis of the corpus callosum in the Kif7-null model.
    evidence:
    - reference: PMID:30445565
      reference_title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
      supports: SUPPORT
      evidence_source: MODEL_ORGANISM
      snippet: >-
        CC agenesis in these mice is associated with specific patterning defects
        of the cortical septum boundary leading to altered distribution of
        guidepost cells required to guide the callosal axons through the
        midline.
      explanation: >-
        The model associates guidepost-cell mispatterning with callosal
        agenesis and identifies failed midline axon guidance as the intervening
        step.
- name: Cerebellar and CNS Malformation
  conforms_to: "ciliopathy_dysfunction#Cerebellar and CNS Malformation"
  description: >-
    Impaired cilium-dependent Hedgehog signaling during neurodevelopment
    produces the hallmark CNS malformations of ACLS: agenesis or hypoplasia of
    the corpus callosum and cerebellar vermis dysgenesis. In a subset of
    patients the cerebellar/brainstem malformation is severe enough to generate
    the molar tooth sign on MRI, overlapping with Joubert syndrome and
    reflecting the shared ciliary Hedgehog mechanism.
  biological_processes:
  - preferred_term: Corpus Callosum Development
    term:
      id: GO:0022038
      label: corpus callosum development
    modifier: ABNORMAL
  - preferred_term: Cerebellum Development
    term:
      id: GO:0021549
      label: cerebellum development
    modifier: ABNORMAL
  cell_types:
  - preferred_term: Neuron
    term:
      id: CL:0000540
      label: neuron
  locations:
  - preferred_term: Corpus Callosum
    term:
      id: UBERON:0002336
      label: corpus callosum
  - preferred_term: Cerebellar Vermis
    term:
      id: UBERON:0004720
      label: cerebellar vermis
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Acrocallosal syndrome (ACLS) is a rare recessive disorder characterised by
      corpus callosum agenesis or hypoplasia, craniofacial dysmorphism,
      duplication of the hallux, postaxial polydactyly, and severe mental
      retardation
    explanation: >-
      Establishes corpus callosum agenesis/hypoplasia as a defining CNS feature
      of ACLS.
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly
      indicated the diagnosis
    explanation: >-
      Documents cerebellar vermis dysgenesis and the molar tooth sign as
      diagnostically informative CNS malformations in ACLS.
  downstream:
  - target: Hypoplasia of Corpus Callosum
    causal_link_type: DIRECT
    description: Abnormal callosal development can produce corpus-callosum hypoplasia.
    evidence:
    - reference: PMID:23125460
      reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        corpus callosum agenesis or hypoplasia, craniofacial dysmorphism,
        duplication of the hallux, postaxial polydactyly
      explanation: >-
        Clinical evidence supports hypoplasia as a direct outcome of abnormal
        callosal development; the cellular route is not inferred from the
        agenesis-only mouse model.
  - target: Cerebellar Vermis Hypoplasia
    causal_link_type: DIRECT
    description: Abnormal cerebellar development produces vermis hypoplasia or dysgenesis.
    evidence:
    - reference: PMID:23125460
      reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly
        indicated the diagnosis
      explanation: >-
        Direct clinical imaging evidence links the CNS-malformation node to
        vermis dysgenesis.
  - target: Molar Tooth Sign
    causal_link_type: DIRECT
    description: Severe cerebellar and brainstem malformation can produce the molar tooth sign.
    evidence:
    - reference: PMID:23125460
      reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly
        indicated the diagnosis
      explanation: >-
        Directly documents the molar-tooth imaging configuration in the
        KIF7-associated spectrum.
  - target: Dandy-Walker Malformation
    causal_link_type: DIRECT
    description: >-
      Severe cerebellar and vermian developmental disruption can produce a
      Dandy-Walker malformation.
    evidence:
    - reference: PMID:29910499
      reference_title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        showed partial callosal agenesis, pachygyria, ventriculomegaly, agenesis
        of cerebellar vermis and Dandy–Walker malformation.
      explanation: >-
        A KIF7-confirmed infant had the Dandy-Walker configuration on MRI.
  - target: Intellectual Disability
    causal_link_type: INDIRECT_UNKNOWN_INTERMEDIATES
    description: >-
      KIF7-associated developmental brain disruption is clinically linked to
      intellectual disability, but the precise causal mediators are not
      resolved and are not reduced to callosal agenesis alone.
    evidence:
    - reference: PMID:29321670
      reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        patients with pathogenic KIF7 variants had more severe and persistent
        facial dysmorphism and in general more severe ID
      explanation: >-
        Supports the clinical association while the downstream cellular route
        remains unresolved.
  - target: Hypotonia
    causal_link_type: INDIRECT_UNKNOWN_INTERMEDIATES
    description: >-
      Developmental CNS dysfunction can manifest as hypotonia, although the
      responsible circuits have not been resolved.
    evidence:
    - reference: PMID:22696705
      reference_title: "Acrocallosal syndrome in a young hypertensive male."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        Except for hypotonia, rest of the central nervous system examination was
        normal.
      explanation: Documents hypotonia in a clinically diagnosed adult case.
  - target: Seizure
    causal_link_type: INDIRECT_UNKNOWN_INTERMEDIATES
    description: >-
      Seizures occur in a subset of KIF7-positive individuals, but the exact
      epileptogenic mechanism is unresolved.
    evidence:
    - reference: PMID:29321670
      reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: "Seizure7/18 (39%)2/19 (11%)"
      explanation: >-
        The pooled KIF7-positive ACLS/Joubert spectrum had seizures in 7 of 18
        individuals with available data.
- name: Limb and Craniofacial Patterning Defect
  description: >-
    KIF7-dependent Hedgehog/GLI3 signaling is required for dosed limb and
    craniofacial patterning. Deregulated GLI output produces polydactyly,
    hallux duplication, syndactyly, cleft palate, and the characteristic
    macrocephalic craniofacial appearance.
  biological_processes:
  - preferred_term: limb morphogenesis
    term:
      id: GO:0035108
      label: limb morphogenesis
    modifier: ABNORMAL
  - preferred_term: palate development
    term:
      id: GO:0060021
      label: roof of mouth development
    modifier: ABNORMAL
  evidence:
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      overlapping features that include polydactyly, brain abnormalities and
      cleft palate
    explanation: >-
      Original KIF7 report supports limb and craniofacial malformations as
      part of the KIF7 ciliopathy spectrum.
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      characterized by postaxial and/or preaxial polydactyly, cutaneous
      syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of the
      corpus callosum, and intellectual disability
    explanation: >-
      Review evidence connects KIF7-associated ACLS to polydactyly,
      syndactyly, macrocephaly, and hypertelorism.
  downstream:
  - target: Postaxial Polydactyly
    causal_link_type: DIRECT
    description: Abnormal limb patterning produces postaxial polydactyly.
    evidence:
    - reference: PMID:26349186
      reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        characterized by postaxial and/or preaxial polydactyly, cutaneous
        syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of
        the corpus callosum, and intellectual disability
      explanation: Supports postaxial polydactyly as an output of this patterning defect.
  - target: Preaxial Polydactyly
    causal_link_type: DIRECT
    description: Abnormal limb patterning produces preaxial polydactyly.
    evidence:
    - reference: PMID:26648833
      reference_title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        postaxial polydactyly of the hands as well as preaxial polydactyly of
        the feet
      explanation: Supports the two region-specific polydactyly outcomes.
  - target: Duplication of the Hallux
    causal_link_type: DIRECT
    description: Preaxial foot patterning disruption produces hallux duplication.
    evidence:
    - reference: PMID:23125460
      reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        corpus callosum agenesis or hypoplasia, craniofacial dysmorphism,
        duplication of the hallux, postaxial polydactyly
      explanation: Supports hallux duplication as a characteristic limb outcome.
  - target: Cutaneous Syndactyly
    causal_link_type: DIRECT
    description: Digital patterning disruption produces soft-tissue syndactyly.
    evidence:
    - reference: PMID:26349186
      reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        characterized by postaxial and/or preaxial polydactyly, cutaneous
        syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of
        the corpus callosum, and intellectual disability
      explanation: Supports cutaneous syndactyly as part of the digital pattern.
  - target: Cleft Palate
    causal_link_type: DIRECT
    description: Midline craniofacial patterning disruption produces cleft palate.
    evidence:
    - reference: PMID:21552264
      reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        overlapping features that include polydactyly, brain abnormalities and
        cleft palate
      explanation: Supports cleft palate within the KIF7 malformation spectrum.
  - target: Macrocephaly
    causal_link_type: INDIRECT_UNKNOWN_INTERMEDIATES
    description: ACLS craniofacial patterning disruption contributes to macrocephaly.
    evidence:
    - reference: PMID:26349186
      reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        characterized by postaxial and/or preaxial polydactyly, cutaneous
        syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of
        the corpus callosum, and intellectual disability
      explanation: Supports macrocephaly as an associated craniofacial outcome.
  - target: Hypertelorism
    causal_link_type: DIRECT
    description: Craniofacial patterning disruption produces hypertelorism.
    evidence:
    - reference: PMID:23125460
      reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        Facial dysmorphism with hypertelorism and prominent forehead in all the
        cases
      explanation: Supports hypertelorism as a recurring craniofacial outcome.
  - target: Prominent Forehead
    causal_link_type: DIRECT
    description: Craniofacial patterning disruption produces a prominent forehead.
    evidence:
    - reference: PMID:23125460
      reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
      supports: SUPPORT
      evidence_source: HUMAN_CLINICAL
      snippet: >-
        Facial dysmorphism with hypertelorism and prominent forehead in all the
        cases
      explanation: Supports prominent forehead as a recurring craniofacial outcome.
phenotypes:
- name: Agenesis of Corpus Callosum
  category: Neurologic
  description: >-
    Complete agenesis of the corpus callosum is a cardinal ACLS feature and
    gives the syndrome its name; partial hypoplasia is represented separately.
  phenotype_term:
    preferred_term: Agenesis of corpus callosum
    term:
      id: HP:0001274
      label: Agenesis of corpus callosum
  evidence:
  - reference: PMID:26648833
    reference_title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Acrocallosal syndrome (ACLS) is a rare autosomal recessive disorder
      characterized by agenesis of the corpus callosum
    explanation: >-
      Corpus callosum agenesis is the eponymous, defining CNS malformation of
      ACLS.
- name: Hypoplasia of Corpus Callosum
  category: Neurologic
  description: >-
    Partial underdevelopment of the corpus callosum is an alternative to
    complete agenesis within the KIF7-related ACLS spectrum.
  phenotype_term:
    preferred_term: Hypoplasia of the corpus callosum
    term:
      id: HP:0002079
      label: Hypoplasia of the corpus callosum
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Acrocallosal syndrome (ACLS) is a rare recessive disorder characterised by
      corpus callosum agenesis or hypoplasia
    explanation: >-
      Explicitly documents hypoplasia as part of the callosal phenotype rather
      than collapsing all cases into complete agenesis.
- name: Postaxial Polydactyly
  category: Skeletal
  description: >-
    Postaxial polydactyly of the hands is one of the two cardinal limb features
    of ACLS, reflecting deregulated Hedgehog/GLI3 anterior-posterior limb
    patterning.
  phenotype_term:
    preferred_term: Postaxial hand polydactyly
    term:
      id: HP:0001162
      label: Postaxial hand polydactyly
  evidence:
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Acrocallosal syndrome is a multiple congenital anomaly disorder
      characterized by postaxial and/or preaxial polydactyly
    explanation: >-
      Postaxial polydactyly is a cardinal limb malformation of ACLS.
- name: Preaxial Polydactyly
  category: Skeletal
  description: >-
    Preaxial polydactyly of the feet, frequently manifesting as duplication of
    the hallux, is characteristic of ACLS.
  phenotype_term:
    preferred_term: Preaxial foot polydactyly
    term:
      id: HP:0001841
      label: Preaxial foot polydactyly
  evidence:
  - reference: PMID:26648833
    reference_title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      postaxial polydactyly of the hands as well as preaxial polydactyly of the
      feet
    explanation: >-
      Preaxial polydactyly of the feet is a recognized cardinal limb feature of
      ACLS.
- name: Duplication of the Hallux
  category: Skeletal
  description: >-
    Duplication of the hallux (great toe) is a distinctive preaxial digital
    malformation in ACLS.
  phenotype_term:
    preferred_term: Duplication of the hallux
    term:
      id: HP:0010066
      label: Duplication of phalanx of hallux
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      corpus callosum agenesis or hypoplasia, craniofacial dysmorphism,
      duplication of the hallux, postaxial polydactyly
    explanation: >-
      Hallux duplication is listed among the defining clinical features of ACLS.
- name: Macrocephaly
  category: Craniofacial
  description: >-
    Macrocephaly (enlarged head) is a consistent craniofacial feature of ACLS.
  phenotype_term:
    preferred_term: Macrocephaly
    term:
      id: HP:0000256
      label: Macrocephaly
  evidence:
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      characterized by postaxial and/or preaxial polydactyly, cutaneous
      syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of the
      corpus callosum, and intellectual disability
    explanation: >-
      Macrocephaly is enumerated among the characteristic features of ACLS.
- name: Hypertelorism
  category: Craniofacial
  frequency: VERY_FREQUENT
  description: >-
    Widely spaced eyes (hypertelorism) are part of the characteristic
    craniofacial dysmorphism of ACLS.
  phenotype_term:
    preferred_term: Hypertelorism
    term:
      id: HP:0000316
      label: Hypertelorism
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Facial dysmorphism with hypertelorism and prominent forehead in all the
      cases
    explanation: >-
      Hypertelorism was present in all cases of this ACLS series, supporting
      VERY_FREQUENT classification.
- name: Prominent Forehead
  category: Craniofacial
  frequency: VERY_FREQUENT
  description: >-
    A prominent (bossed) forehead is a consistent craniofacial feature of ACLS.
  phenotype_term:
    preferred_term: Prominent forehead
    term:
      id: HP:0011220
      label: Prominent forehead
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Facial dysmorphism with hypertelorism and prominent forehead in all the
      cases
    explanation: >-
      Prominent forehead was present in all cases of this ACLS series, supporting
      VERY_FREQUENT classification.
- name: Intellectual Disability
  category: Neurodevelopmental
  description: >-
    Intellectual disability, frequently severe, is a core neurodevelopmental
    feature of ACLS.
  phenotype_term:
    preferred_term: Intellectual disability
    term:
      id: HP:0001249
      label: Intellectual disability
  evidence:
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      absence or hypoplasia of the corpus callosum, and intellectual disability
    explanation: >-
      Intellectual disability is a defining neurodevelopmental feature of ACLS.
- name: Hypotonia
  category: Neurologic
  description: >-
    Reduced muscle tone is a recognized neurologic manifestation and can
    compound early motor delay.
  phenotype_term:
    preferred_term: Hypotonia
    term:
      id: HP:0001252
      label: Hypotonia
  evidence:
  - reference: PMID:22696705
    reference_title: "Acrocallosal syndrome in a young hypertensive male."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Except for hypotonia, rest of the central nervous system examination was
      normal.
    explanation: Documents hypotonia in a clinically diagnosed adult case.
- name: Seizure
  category: Neurologic
  frequency: FREQUENT
  description: >-
    Seizures occur in a substantial minority of individuals across the
    KIF7-positive ACLS/Joubert-spectrum literature.
  phenotype_term:
    preferred_term: Seizure
    term:
      id: HP:0001250
      label: Seizure
  evidence:
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "Seizure7/18 (39%)2/19 (11%)"
    explanation: >-
      The KIF7-positive ACLS/Joubert spectrum had seizures in 7 of 18
      individuals with available data, supporting the FREQUENT band.
- name: Cerebellar Vermis Hypoplasia
  category: Neurologic
  description: >-
    Cerebellar vermis dysgenesis/hypoplasia, sometimes severe enough to produce
    the molar tooth sign, occurs in ACLS and overlaps with Joubert syndrome.
  phenotype_term:
    preferred_term: Cerebellar vermis hypoplasia
    term:
      id: HP:0001320
      label: Cerebellar vermis hypoplasia
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly
      indicated the diagnosis
    explanation: >-
      Cerebellar vermis dysgenesis is a CNS malformation supporting the
      diagnosis of ACLS.
- name: Molar Tooth Sign
  category: Neurologic
  description: >-
    The molar tooth sign on MRI (deepened interpeduncular fossa with elongated,
    thickened superior cerebellar peduncles) can occur in ACLS, reflecting the
    shared ciliary Hedgehog mechanism with Joubert syndrome.
  phenotype_term:
    preferred_term: Molar tooth sign on MRI
    term:
      id: HP:0002419
      label: Molar tooth sign on MRI
  evidence:
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      They present with features like molar tooth sign and hyperventilation
      that are not very typical in ACLS, but do occur in other ciliopathies
    explanation: >-
      The molar tooth sign is described as "not very typical" in ACLS, supporting
      its presence in a variable subset without assigning a frequency from
      qualitative wording.
- name: Dandy-Walker Malformation
  category: Neurologic
  description: >-
    Dandy-Walker malformation is a variable cerebellar/posterior-fossa feature
    documented in the KIF7-related ACLS spectrum; it is not present in every
    affected individual.
  phenotype_term:
    preferred_term: Dandy-Walker malformation
    term:
      id: HP:0001305
      label: Dandy-Walker malformation
  evidence:
  - reference: PMID:29910499
    reference_title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      showed partial callosal agenesis, pachygyria, ventriculomegaly, agenesis
      of cerebellar vermis and Dandy–Walker malformation.
    explanation: >-
      A KIF7-confirmed infant had Dandy-Walker malformation on MRI, supporting
      inclusion without assigning a population frequency.
- name: Cleft Palate
  category: Craniofacial
  description: >-
    Cleft palate occurs in a subset of ACLS cases and is also a feature of the
    allelic hydrolethalus syndrome, reflecting shared deregulation of midline
    craniofacial Hedgehog patterning.
  phenotype_term:
    preferred_term: Cleft palate
    term:
      id: HP:0000175
      label: Cleft palate
  evidence:
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      polydactyly, brain abnormalities and cleft palate
    explanation: >-
      Cleft palate is listed among the overlapping malformation features of
      hydrolethalus and acrocallosal syndromes in the original KIF7 discovery paper.
- name: Cutaneous Syndactyly
  category: Skeletal
  description: >-
    Cutaneous (soft-tissue) syndactyly of the digits is a recognized digital
    malformation in ACLS, co-occurring with polydactyly.
  phenotype_term:
    preferred_term: Cutaneous syndactyly
    term:
      id: HP:0012725
      label: Cutaneous syndactyly
  evidence:
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      characterized by postaxial and/or preaxial polydactyly, cutaneous
      syndactyly, macrocephaly, widely spaced eyes, absence or hypoplasia of the
      corpus callosum, and intellectual disability
    explanation: >-
      Cutaneous syndactyly is enumerated among the characteristic limb features
      of ACLS in this KIF7 syndromology review.
imaging_findings:
- name: Corpus Callosum Agenesis or Hypoplasia on MRI
  modality: MRI
  imaging_finding_term:
    preferred_term: Abnormal corpus callosum morphology
    term:
      id: HP:0001273
      label: Abnormal corpus callosum morphology
  phenotype_term:
    preferred_term: Abnormal corpus callosum morphology
    term:
      id: HP:0001273
      label: Abnormal corpus callosum morphology
  located_in:
    preferred_term: Corpus Callosum
    term:
      id: UBERON:0002336
      label: corpus callosum
  description: >-
    Brain MRI demonstrates complete agenesis or partial hypoplasia of the
    corpus callosum. This is a cardinal diagnostic component of the syndrome,
    although molecular confirmation is required for KIF7-specific diagnosis.
  diagnostic: true
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      corpus callosum agenesis or hypoplasia, craniofacial dysmorphism,
      duplication of the hallux, postaxial polydactyly
    explanation: >-
      Establishes callosal agenesis or hypoplasia as a defining structural
      neuroimaging finding.
  - reference: PMID:29910499
    reference_title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: Magnetic resonance imaging (MRI) of the brain
    explanation: >-
      Establishes MRI as the modality used for the structural brain findings in
      this KIF7-confirmed case.
  - reference: PMID:29910499
    reference_title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      showed partial callosal agenesis, pachygyria, ventriculomegaly, agenesis
      of cerebellar vermis and Dandy–Walker malformation.
    explanation: >-
      The same MRI sentence documents partial callosal agenesis in a
      KIF7-confirmed case.
- name: Molar Tooth Sign on MRI
  modality: MRI
  imaging_finding_term:
    preferred_term: Molar tooth sign on MRI
    term:
      id: HP:0002419
      label: Molar tooth sign on MRI
  phenotype_term:
    preferred_term: Molar tooth sign on MRI
    term:
      id: HP:0002419
      label: Molar tooth sign on MRI
  located_in:
    preferred_term: Brainstem
    term:
      id: UBERON:0002298
      label: brainstem
  description: >-
    Vermis dysgenesis with associated brainstem configuration can create the
    molar tooth sign. It occurs in a variable subset and signals overlap with
    the KIF7-related Joubert spectrum rather than defining every ACLS case.
  diagnostic: false
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      vermis dysgenesis with brainstem anomalies (molar tooth sign), strongly
      indicated the diagnosis
    explanation: Documents the molar tooth configuration on brain imaging.
- name: Olfactory Bulb and Tract Abnormalities on Neuroimaging
  located_in:
    preferred_term: Olfactory Bulb
    term:
      id: UBERON:0002264
      label: olfactory bulb
  description: >-
    Olfactory-bulb and olfactory-tract abnormalities were reported on
    neuroimaging in one child with a KIF7-confirmed acrocallosal phenotype. The
    available abstract does not isolate the modality for this finding, so none
    is asserted here. This is an emerging single-case extension, not a defining
    or frequency-estimated imaging feature.
  diagnostic: false
  context: Single KIF7-confirmed case report
  evidence:
  - reference: PMID:31399769
    reference_title: "Olfactory bulb and olfactory tract abnormalities in acrocallosal syndrome and Greig cephalopolysyndactyly syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      We describe association of olfactory bulb and olfactory tract
      abnormalities in a child with acrocallosal syndrome caused by kinesin
      family membrane 7 (KIF7) mutation in sonic hedgehog pathway.
    explanation: >-
      Molecularly confirmed single-case evidence supports this explicitly
      qualified imaging extension.
genetic:
- name: Biallelic KIF7 Variants
  association: Causative
  relationship_type: CAUSATIVE
  variant_origin: GERMLINE
  gene_term:
    preferred_term: KIF7
    term:
      id: hgnc:30497
      label: KIF7
  notes: >-
    Biallelic pathogenic variants in KIF7 (15q26.1) cause the autosomal
    recessive form modeled here. Truncating and splice-disrupting alleles are
    well established; biallelic missense substitutions have also been reported,
    but individual missense findings require variant-level interpretation.
    Allelic KIF7 disease spans the more severe hydrolethalus syndrome 2 and
    Joubert-spectrum presentations. Rare de novo GLI3-related acrocallosal
    phenocopies are handled as a differential, not as a second causal gene in
    this KIF7-scoped entry.
  evidence:
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      we report mutations in KIF7 in individuals with hydrolethalus and
      acrocallosal syndromes, two multiple malformation disorders with
      overlapping features that include polydactyly, brain abnormalities and
      cleft palate
    explanation: >-
      Original report identifying KIF7 as the causative gene for both ACLS and
      the allelic hydrolethalus syndrome.
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Seven mutations were identified at the KIF7 locus in these five cases, six
      of which are novel
    explanation: >-
      Mutation series confirming KIF7 as the recurrent disease locus and
      expanding the allelic spectrum of ACLS.
  - reference: PMID:26648833
    reference_title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Mutations in the KIF7 gene, encoding a molecule within the Sonic hedgehog
      (SHH) pathway, have been identified as causative for ACLS but also for the
      fatal hydrolethalus syndrome and some cases of Joubert syndrome
    explanation: >-
      Confirms KIF7 causation of ACLS and the allelic relationship with
      hydrolethalus and Joubert syndromes.
  - reference: PMID:26174511
    reference_title: "Novel KIF7 missense substitutions in two patients presenting with multiple malformations and features of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      We present two children who both had two missense mutations in the Kinesin
      Family Member 7 (KIF7) gene.
    explanation: >-
      Supports only that biallelic missense substitutions have been reported;
      the paper itself was tentative and does not establish every reported
      missense allele as pathogenic.
diagnosis:
- name: Clinical and Molecular Diagnosis
  description: >-
    Acrocallosal syndrome is suspected from the combination of corpus callosum
    agenesis/hypoplasia, craniofacial dysmorphism (macrocephaly, prominent
    forehead, hypertelorism), and polydactyly, and is confirmed by
    identification of biallelic KIF7 pathogenic variants. Because expressivity
    is variable, KIF7 testing is warranted in patients with suggestive
    craniofacial features even when polydactyly or callosal anomalies are
    absent. If targeted KIF7 testing is negative, a broader ciliopathy/
    polydactyly panel or exome analysis can evaluate phenocopies involving
    CPLANE1/C5orf42, GLI3, SHH, and other developmental-pathway genes.
  diagnosis_term:
    preferred_term: molecular genetic testing
    term:
      id: NCIT:C19770
      label: Molecular Analysis
  evidence:
  - reference: PMID:23125460
    reference_title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      KIF7 should be tested in less typical patients in whom craniofacial
      features are suggestive of ACLS
    explanation: >-
      Defines the molecular diagnostic recommendation for ACLS, including in
      atypical presentations.
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Genetic analysis in our cohort of patients with craniofacial appearance
      fitting into the ACLS clinical spectrum and variable additional
      ciliopathy features revealed (likely) pathogenic variants not only in
      KIF7, but also secondly in C5orf42
    explanation: >-
      Demonstrates locus heterogeneity in ACLS-like presentations and supports
      broader sequencing when KIF7 testing is unrevealing.
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      In addition, in one patient we found a heterozygous de novo frameshift
      variant in SHH, the most common cause of non-chromosomal holoprosencephaly
      (HPE)
    explanation: >-
      Supports inclusion of SHH-related phenocopies in a broader negative-KIF7
      diagnostic workup.
- name: Prenatal Ultrasound Assessment
  description: >-
    In an at-risk pregnancy, second-trimester ultrasound can identify
    polydactyly and major central-nervous-system malformations, but variable
    expressivity means a normal scan does not exclude KIF7-related disease.
  diagnosis_term:
    preferred_term: clinical imaging procedure
    term:
      id: NCIT:C17369
      label: Imaging Procedure
  evidence:
  - reference: PMID:24949054
    reference_title: "The acrocallosal syndrome in a neonate with further widening of phenotypic expression."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Polydactyly, and central nervous system malformations can be detected by
      ultrasonography in the second trimester, but due to variability of
      presentation, prenatal diagnosis may not always be possible.
    explanation: >-
      Supports both the detectable prenatal features and the limitation of
      ultrasound caused by variable presentation.
differential_diagnoses:
- name: KIF7-Related Hydrolethalus Syndrome 2
  disease_term:
    preferred_term: hydrolethalus syndrome 2
    term:
      id: MONDO:0013585
      label: hydrolethalus syndrome 2
  description: >-
    Hydrolethalus syndrome 2 is the KIF7-related, severe fetal/lethal end of the
    allelic spectrum and shares polydactyly, brain abnormalities, and cleft
    palate with ACLS. Hydrolethalus syndrome is genetically heterogeneous, so
    this statement does not assign every hydrolethalus presentation to KIF7.
  distinguishing_features:
  - Fetal or perinatal lethality and more severe multisystem malformations favor hydrolethalus syndrome 2 over ACLS.
  evidence:
  - reference: PMID:21552264
    reference_title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      hydrolethalus and acrocallosal syndromes, two multiple malformation
      disorders with overlapping features that include polydactyly, brain
      abnormalities and cleft palate
    explanation: >-
      Establishes hydrolethalus syndrome as the allelic, phenotypically
      overlapping disorder distinguishing the severe end of the KIF7 spectrum.
- name: Joubert Syndrome
  disease_term:
    preferred_term: Joubert syndrome
    term:
      id: MONDO:0018772
      label: Joubert syndrome
  description: >-
    Joubert syndrome shares the molar tooth sign and is caused in a subset of
    cases by KIF7 mutations; distinguished by the predominance of the
    cerebellar/brainstem malformation and characteristic abnormal eye movements
    and breathing dysregulation.
  distinguishing_features:
  - A predominant molar-tooth imaging phenotype with oculomotor apraxia or breathing dysregulation favors a Joubert-spectrum diagnosis over classic ACLS.
  evidence:
  - reference: PMID:26648833
    reference_title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      have been identified as causative for ACLS but also for the fatal
      hydrolethalus syndrome and some cases of Joubert syndrome
    explanation: >-
      Documents the KIF7 allelic overlap between ACLS and Joubert syndrome,
      relevant to differential diagnosis.
  - reference: PMID:26349186
    reference_title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      They present with features like molar tooth sign and hyperventilation
      that are not very typical in ACLS, but do occur in other ciliopathies
    explanation: >-
      Supports the molar-tooth and breathing-regulation distinction from
      classic ACLS.
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      none of the KIF7 patients but all the C5orf42 patients in our cohort
      presented with ocular motor apraxia, which is commonly associated with
      the MTS in JBTS patients
    explanation: >-
      Supports oculomotor apraxia as a discriminator in ACLS-like versus
      Joubert-spectrum presentations while preserving gene-specific context.
- name: CPLANE1-Related Joubert Syndrome 17
  disease_term:
    preferred_term: Joubert syndrome 17
    term:
      id: MONDO:0013824
      label: Joubert syndrome 17
  description: >-
    Biallelic CPLANE1/C5orf42-related Joubert syndrome 17 can closely resemble
    ACLS through macrocephaly, hypertelorism, polydactyly, developmental
    impairment, and a molar tooth/vermis phenotype.
  distinguishing_features:
  - Near-constant molar tooth sign or vermis hypoplasia and ocular motor apraxia favor CPLANE1-related Joubert syndrome 17.
  - Corpus-callosum abnormality is more prominent across the KIF7-positive spectrum.
  evidence:
  - reference: PMID:29321670
    reference_title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      C5orf42 (likely) pathogenic variants are nearly invariably associated to
      the MTS/vermis hypoplasia and less often to corpus callosum abnormalities,
      while it is the opposite for KIF7.
    explanation: >-
      Establishes the overlapping phenotype and the clinical discriminators of
      CPLANE1/C5orf42-related disease.
- name: GLI3-Related Greig-Acrocallosal Spectrum
  disease_term:
    preferred_term: Greig cephalopolysyndactyly syndrome
    term:
      id: MONDO:0008287
      label: Greig cephalopolysyndactyly syndrome
  description: >-
    Rare de novo GLI3 variants have produced an acrocallosal phenotype, and
    GLI3 disorders such as Greig cephalopolysyndactyly overlap through
    macrocephaly, hypertelorism, and polysyndactyly. This is modeled as a
    dominant phenocopy rather than broadening the focal KIF7 entry.
  distinguishing_features:
  - A heterozygous or de novo GLI3 variant supports a dominant GLI3-spectrum diagnosis rather than autosomal recessive KIF7-related ACLS.
  evidence:
  - reference: PMID:23633388
    reference_title: "A de novo GLI3 mutation in a patient with acrocallosal syndrome."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      These data confirm that mutations in GLI3 are a cause of the acrocallosal
      phenotype.
    explanation: >-
      Supports the explicit GLI3 phenocopy boundary and its placement in the
      differential rather than the focal KIF7 genetic model.
treatments:
- name: Supportive and Multidisciplinary Care
  description: >-
    Management is symptom-directed and multidisciplinary. Early physical,
    occupational, developmental, and communication support addresses hypotonia
    and developmental disability; seizures are treated with standard
    antiseizure therapy when present. Available evidence is case-report and
    clinical-review level rather than ACLS-specific comparative trials.
  action_category: THERAPEUTIC
  treatment_term:
    preferred_term: supportive care
    term:
      id: NCIT:C15747
      label: Supportive Care
  target_phenotypes:
  - preferred_term: Hypotonia
    term:
      id: HP:0001252
      label: Hypotonia
  - preferred_term: Intellectual disability
    term:
      id: HP:0001249
      label: Intellectual disability
  - preferred_term: Seizure
    term:
      id: HP:0001250
      label: Seizure
  evidence:
  - reference: PMID:22696705
    reference_title: "Acrocallosal syndrome in a young hypertensive male."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Treatment includes physical therapy in infancy which may assist in the
      development of motor skills and muscle tone.
    explanation: Supports early physical therapy for hypotonia and motor delay.
  - reference: PMID:22696705
    reference_title: "Acrocallosal syndrome in a young hypertensive male."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Antiepileptic therapy for seizure disorder and antihypertensives for
      renoparenchymal hypertension are to be given.
    explanation: Supports symptom-directed antiseizure treatment when seizures occur.
  - reference: PMID:29910499
    reference_title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      At present, the child is referred to a neurodevelopment specialist, for
      neurodevelopment assessment and occupational therapist for infant
      stimulation program.
    explanation: >-
      A genetically confirmed case documents specialist developmental follow-up
      and occupational-therapy referral.
- name: Surgical Correction of Polydactyly and Malformations
  description: >-
    Individually indicated surgery may remove functionally limiting extra
    digits, release syndactyly, or repair orofacial and other structural
    malformations. These procedures treat local manifestations rather than the
    underlying ciliary signaling defect.
  action_category: THERAPEUTIC
  therapeutic_modality: SURGERY
  treatment_term:
    preferred_term: surgical procedure
    term:
      id: NCIT:C15329
      label: Surgical Procedure
  target_phenotypes:
  - preferred_term: Postaxial hand polydactyly
    term:
      id: HP:0001162
      label: Postaxial hand polydactyly
  - preferred_term: Preaxial foot polydactyly
    term:
      id: HP:0001841
      label: Preaxial foot polydactyly
  - preferred_term: Cutaneous syndactyly
    term:
      id: HP:0012725
      label: Cutaneous syndactyly
  - preferred_term: Cleft palate
    term:
      id: HP:0000175
      label: Cleft palate
  evidence:
  - reference: PMID:22696705
    reference_title: "Acrocallosal syndrome in a young hypertensive male."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Surgery to remove extra fingers and toes and release fused fingers may
      improve movement and grasp, may assist in walking and the comfort of
      footwear.
    explanation: >-
      Supports symptom-directed surgery for polydactyly and syndactyly while
      making no disease-modifying claim.
  - reference: PMID:29910499
    reference_title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      She underwent Nissen fundoplication at 4 months of age, cleft lip repair
      at 6 months age and cleft palate repair at 18 months of age using the same
      technique.
    explanation: >-
      A KIF7-confirmed case documents actual surgical repair of associated
      structural manifestations, including cleft palate.
- name: Genetic Counseling
  description: >-
    When biallelic KIF7 disease is confirmed, parents should be counseled about
    autosomal recessive inheritance and a 25% recurrence risk in each pregnancy.
    Prenatal ultrasound can detect polydactyly and major CNS malformations but
    cannot exclude disease because presentation is variable. This recurrence
    statement does not apply to a rare de novo GLI3 phenocopy.
  action_category: COUNSELING_INFORMATIONAL
  treatment_term:
    preferred_term: Genetic Counseling
    term:
      id: NCIT:C15240
      label: Genetic Counseling
  evidence:
  - reference: PMID:24949054
    reference_title: "The acrocallosal syndrome in a neonate with further widening of phenotypic expression."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      As the mode of inheritance of ACLS is autosomal recessive, the risk of
      recurrence is 25%. Genetic counselling is of prime importance
    explanation: >-
      Directly supports recurrence-risk counseling for the autosomal recessive
      KIF7 form.
  - reference: PMID:24949054
    reference_title: "The acrocallosal syndrome in a neonate with further widening of phenotypic expression."
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: >-
      Polydactyly, and central nervous system malformations can be detected by
      ultrasonography in the second trimester, but due to variability of
      presentation, prenatal diagnosis may not always be possible.
    explanation: Supports balanced counseling about prenatal ultrasound capabilities and limits.
references:
- reference: PMID:21552264
  title: "KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes."
- reference: PMID:22696705
  title: "Acrocallosal syndrome in a young hypertensive male."
- reference: PMID:23125460
  title: "Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome."
- reference: PMID:23633388
  title: "A de novo GLI3 mutation in a patient with acrocallosal syndrome."
- reference: PMID:24949054
  title: "The acrocallosal syndrome in a neonate with further widening of phenotypic expression."
- reference: PMID:26174511
  title: "Novel KIF7 missense substitutions in two patients presenting with multiple malformations and features of acrocallosal syndrome."
- reference: PMID:26349186
  title: "Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology."
- reference: PMID:26648833
  title: "Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature."
- reference: PMID:29321670
  title: "Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling."
- reference: PMID:29910499
  title: "Anaesthetising an infant with acrocallosal syndrome: An unusual case."
- reference: PMID:30445565
  title: "Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice."
- reference: PMID:31399769
  title: "Olfactory bulb and olfactory tract abnormalities in acrocallosal syndrome and Greig cephalopolysyndactyly syndrome."
- reference: PMID:34705483
  title: "Hedgehog-induced ciliary trafficking of kinesin-4 motor KIF7 requires intraflagellar transport but not KIF7's microtubule binding."
📚

References & Deep Research

References

13
KIF7 mutations cause fetal hydrolethalus and acrocallosal syndromes.
No top-level findings curated for this source.
Acrocallosal syndrome in a young hypertensive male.
No top-level findings curated for this source.
Novel KIF7 mutations extend the phenotypic spectrum of acrocallosal syndrome.
No top-level findings curated for this source.
A de novo GLI3 mutation in a patient with acrocallosal syndrome.
No top-level findings curated for this source.
The acrocallosal syndrome in a neonate with further widening of phenotypic expression.
No top-level findings curated for this source.
Novel KIF7 missense substitutions in two patients presenting with multiple malformations and features of acrocallosal syndrome.
No top-level findings curated for this source.
Molar tooth sign and acrocallosal syndrome--a report on a Polish family and review of KIF7 syndromology.
No top-level findings curated for this source.
Novel KIF7 Mutation in a Tunisian Boy with Acrocallosal Syndrome: Case Report and Review of the Literature.
No top-level findings curated for this source.
Clinical and experimental evidence suggest a link between KIF7 and C5orf42-related ciliopathies through Sonic Hedgehog signaling.
No top-level findings curated for this source.
Anaesthetising an infant with acrocallosal syndrome: An unusual case.
No top-level findings curated for this source.
Altered GLI3 and FGF8 signaling underlies acrocallosal syndrome phenotypes in Kif7 depleted mice.
No top-level findings curated for this source.
Olfactory bulb and olfactory tract abnormalities in acrocallosal syndrome and Greig cephalopolysyndactyly syndrome.
No top-level findings curated for this source.
Hedgehog-induced ciliary trafficking of kinesin-4 motor KIF7 requires intraflagellar transport but not KIF7's microtubule binding.
No top-level findings curated for this source.