A

Disease A

Slug:Asthma
B

Disease B

Slug:Chronic_Obstructive_Pulmonary_Disease
G

Causal Mechanism Graphs

Asthma

graph LR
    Chest_Tightness["Chest Tightness"]
    Exercise_Intolerance["Exercise Intolerance"]
    Sleep_Disturbance["Sleep Disturbance"]
    Type_2_Immune_Response___Th2_Signaling["Type 2 Immune Response / Th2 Signaling"]
    IL4["IL4"]
    Wheezing["Wheezing"]
    IL13["IL13"]
    Airway_Inflammation["Airway Inflammation"]
    Early_Life_Rhinovirus_Wheezing_Illness_and_Asthma_Inception["Early-Life Rhinovirus Wheezing Illness and Asthma Inception"]
    Rapid_Breathing["Rapid Breathing"]
    Respiratory_Distress["Respiratory Distress"]
    STAT3["STAT3"]
    Coughing["Coughing"]
    Bronchoconstriction["Bronchoconstriction"]
    Reduced_Exercise_Tolerance["Reduced Exercise Tolerance"]
    CDHR3["CDHR3"]
    Anxiety["Anxiety"]
    Airway_Remodeling["Airway Remodeling"]
    Mucus_Overproduction["Mucus Overproduction"]
    ADAM33["ADAM33"]
    House_Dust_Mite_Allergen_Protease_Induced_Epithelial_Oxidant_Signaling["House Dust Mite Allergen Protease-Induced Epithelial Oxidant Signaling"]
    Fatigue["Fatigue"]
    Cyanosis["Cyanosis"]
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation["SIRT1-Mediated NAD+ Signaling and Protective Deacetylation"]

    Airway_Inflammation --> Bronchoconstriction
    Type_2_Immune_Response___Th2_Signaling --> Airway_Inflammation
    Type_2_Immune_Response___Th2_Signaling --> Mucus_Overproduction
    Bronchoconstriction --> Wheezing
    Bronchoconstriction --> Chest_Tightness
    Bronchoconstriction --> Rapid_Breathing
    Bronchoconstriction --> Respiratory_Distress
    Bronchoconstriction --> Cyanosis
    Bronchoconstriction --> Exercise_Intolerance
    Bronchoconstriction --> Reduced_Exercise_Tolerance
    Bronchoconstriction --> Fatigue
    Bronchoconstriction --> Anxiety
    Mucus_Overproduction --> Coughing
    Mucus_Overproduction --> Sleep_Disturbance
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Type_2_Immune_Response___Th2_Signaling
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Mucus_Overproduction
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Airway_Remodeling
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Bronchoconstriction
    Early_Life_Rhinovirus_Wheezing_Illness_and_Asthma_Inception --> Type_2_Immune_Response___Th2_Signaling
    House_Dust_Mite_Allergen_Protease_Induced_Epithelial_Oxidant_Signaling --> Airway_Inflammation
    Wheezing --> Respiratory_Distress
    Wheezing --> Reduced_Exercise_Tolerance
    IL4 --> Type_2_Immune_Response___Th2_Signaling
    IL13 --> Type_2_Immune_Response___Th2_Signaling
    ADAM33 --> Airway_Remodeling
    STAT3 --> SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation
    CDHR3 --> Early_Life_Rhinovirus_Wheezing_Illness_and_Asthma_Inception

    style Chest_Tightness fill:#fef3c7
    style Exercise_Intolerance fill:#fef3c7
    style Sleep_Disturbance fill:#fef3c7
    style Type_2_Immune_Response___Th2_Signaling fill:#dbeafe
    style IL4 fill:#f3e8ff
    style Wheezing fill:#fef3c7
    style IL13 fill:#f3e8ff
    style Airway_Inflammation fill:#dbeafe
    style Early_Life_Rhinovirus_Wheezing_Illness_and_Asthma_Inception fill:#dbeafe
    style Rapid_Breathing fill:#fef3c7
    style Respiratory_Distress fill:#fef3c7
    style STAT3 fill:#f3e8ff
    style Coughing fill:#fef3c7
    style Bronchoconstriction fill:#dbeafe
    style Reduced_Exercise_Tolerance fill:#fef3c7
    style CDHR3 fill:#f3e8ff
    style Anxiety fill:#fef3c7
    style Airway_Remodeling fill:#dbeafe
    style Mucus_Overproduction fill:#dbeafe
    style ADAM33 fill:#f3e8ff
    style House_Dust_Mite_Allergen_Protease_Induced_Epithelial_Oxidant_Signaling fill:#dbeafe
    style Fatigue fill:#fef3c7
    style Cyanosis fill:#fef3c7
    style SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation fill:#dbeafe

Chronic Obstructive Pulmonary Disease

graph LR
    Alveolar_Destruction["Alveolar Destruction"]
    Progressive_Respiratory_Impairment["Progressive Respiratory Impairment"]
    Exercise_Intolerance["Exercise Intolerance"]
    Respiratory_Failure["Respiratory Failure"]
    Hypoxemia["Hypoxemia"]
    Oxidative_Stress_and_Mitochondrial_Dysfunction["Oxidative Stress and Mitochondrial Dysfunction"]
    Hypercapnia["Hypercapnia"]
    Airflow_Limitation["Airflow Limitation"]
    NFE2L2["NFE2L2"]
    Forced_Expiratory_Volume_in_1_Second_FEV1["Forced Expiratory Volume in 1 Second (FEV1)"]
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation["SIRT1-Mediated NAD+ Signaling and Protective Deacetylation"]
    Chronic_Inflammation["Chronic Inflammation"]
    Cellular_Senescence["Cellular Senescence"]
    Dyspnea["Dyspnea"]

    Airflow_Limitation -.-> Progressive_Respiratory_Impairment
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Chronic_Inflammation
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Cellular_Senescence
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Alveolar_Destruction
    SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation --> Oxidative_Stress_and_Mitochondrial_Dysfunction
    Dyspnea --> Exercise_Intolerance
    Respiratory_Failure --> Hypoxemia
    Respiratory_Failure --> Hypercapnia
    Airflow_Limitation -.-> Forced_Expiratory_Volume_in_1_Second_FEV1
    NFE2L2 --> SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation

    style Alveolar_Destruction fill:#dbeafe
    style Progressive_Respiratory_Impairment fill:#fee2e2,stroke:#dc2626,stroke-dasharray: 5 5
    style Exercise_Intolerance fill:#fef3c7
    style Respiratory_Failure fill:#fef3c7
    style Hypoxemia fill:#fef3c7
    style Oxidative_Stress_and_Mitochondrial_Dysfunction fill:#dbeafe
    style Hypercapnia fill:#fef3c7
    style Airflow_Limitation fill:#dbeafe
    style NFE2L2 fill:#f3e8ff
    style Forced_Expiratory_Volume_in_1_Second_FEV1 fill:#e0e7ff
    style SIRT1_Mediated_NAD+_Signaling_and_Protective_Deacetylation fill:#dbeafe
    style Chronic_Inflammation fill:#dbeafe
    style Cellular_Senescence fill:#dbeafe
    style Dyspnea fill:#fef3c7
S

Association Signals

Signal 1
ICEES EHR_COHORT_ASSOCIATION UNKNOWN
Population:ICEES KG 8-20-2024, UNC Health primary-ciliary-dyskinesia (PCD) cohort (condition-specific base population), patient-level chi-square contingency.
Mapping notes:ICEES reports this pair as a positive correlation. Chi-square is not multiple-testing corrected and is conditioned on the PCD base population, so it corroborates rather than establishes the association. The row quoted is the strongest PCD cohort-year (2016).
Temporal: A before B: , B before A: , Same time:
CHI_SQUARE: 168.58533016733276
CI: -
p: 1.5071340388291068e-38
FDR:
ICEES PCD 2016 cohort co-occurrence of asthma and COPD (N=5688); strong, highly significant positive association.
Source: OTHER
"PCD_UNC_patient_2016_v6_binned_deidentified | 168.58533016733276 | 1 | 1.5071340388291068e-38 | 5688"
ICEES UNC EHR cohort shows a strong, highly significant asthma-COPD co-occurrence in the 2016 PCD cohort.
H

Hypotheses

Hypothesis: chronic airway inflammation and remodeling shared between asthma and COPD produce an overlap phenotype (ACO); long-standing asthma with incompletely reversible airflow limitation can progress toward, or be reclassified as, fixed obstruction characteristic of COPD.
PMID:30461532 (SUPPORT)
Source: HUMAN_CLINICAL
"Asthma and chronic obstructive pulmonary disease (COPD) are common diseases that often overlap."
Review establishes asthma and COPD as frequently overlapping diseases, the clinical basis of the ACO comorbidity signal.
PMID:30461532 (SUPPORT)
Source: HUMAN_CLINICAL
"Epidemiological studies reveal that ACO patients have generally an increased burden of illness and healthcare use in addition to poorer quality of life (QoL) compared with asthma and higher or equal to COPD."
Supports the clinical significance of the overlap: ACO carries greater illness burden and healthcare use than asthma alone.
Pathophysiology:
Shared airway inflammation and fixed airflow obstruction: Overlapping inflammatory airway disease in ACO can lead to progressive, incompletely reversible airflow limitation, blurring the boundary between an asthma and a COPD diagnosis.
Biological processes:
PMID:30461532 (PARTIAL)
Source: HUMAN_CLINICAL
"The term asthma-COPD overlap (ACO) has been used to define this entity but there remain several speculations on its exact definition, impact, pathophysiology, clinical features, and management."
Notes that ACO pathophysiology and definition remain incompletely resolved, supporting the cautious CANDIDATE framing.
Y

Raw YAML

Show YAML
name: com_Asthma__Chronic_Obstructive_Pulmonary_Disease
creation_date: '2026-06-26T00:00:00Z'
curation_status: CANDIDATE
notes: >-
  Asthma-COPD overlap (ACO): a well-recognized clinical entity in which features
  of both diseases coexist, with higher illness burden and healthcare use than
  either alone. Surfaced as the strongest positive both-in-dismech ICEES EHR
  signal (UNC PCD cohort, chi-square=168.6, p=1.5e-38) and corroborated by ACO
  review literature. Directionality is left UNKNOWN: ACO encompasses both
  asthma-predominant and COPD-predominant trajectories, and definitions vary
  across studies, though long-standing asthma is a recognized risk factor for
  later fixed airflow obstruction.

disease_a:
  slug: Asthma
  preferred_term: asthma
  term:
    id: MONDO:0004979
    label: asthma

disease_b:
  slug: Chronic_Obstructive_Pulmonary_Disease
  preferred_term: chronic obstructive pulmonary disease
  term:
    id: MONDO:0005002
    label: chronic obstructive pulmonary disease

directionality: UNKNOWN

hypotheses:
- description: >-
    Hypothesis: chronic airway inflammation and remodeling shared between asthma
    and COPD produce an overlap phenotype (ACO); long-standing asthma with
    incompletely reversible airflow limitation can progress toward, or be
    reclassified as, fixed obstruction characteristic of COPD.
  evidence:
  - reference: PMID:30461532
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "Asthma and chronic obstructive pulmonary disease (COPD) are common diseases that often overlap."
    explanation: >-
      Review establishes asthma and COPD as frequently overlapping diseases,
      the clinical basis of the ACO comorbidity signal.
  - reference: PMID:30461532
    supports: SUPPORT
    evidence_source: HUMAN_CLINICAL
    snippet: "Epidemiological studies reveal that ACO patients have generally an increased burden of illness and healthcare use in addition to poorer quality of life (QoL) compared with asthma and higher or equal to COPD."
    explanation: >-
      Supports the clinical significance of the overlap: ACO carries greater
      illness burden and healthcare use than asthma alone.
  pathophysiology:
  - name: Shared airway inflammation and fixed airflow obstruction
    description: >-
      Overlapping inflammatory airway disease in ACO can lead to progressive,
      incompletely reversible airflow limitation, blurring the boundary between
      an asthma and a COPD diagnosis.
    biological_processes:
    - preferred_term: inflammatory response
      term:
        id: GO:0006954
        label: inflammatory response
    evidence:
    - reference: PMID:30461532
      supports: PARTIAL
      evidence_source: HUMAN_CLINICAL
      snippet: "The term asthma-COPD overlap (ACO) has been used to define this entity but there remain several speculations on its exact definition, impact, pathophysiology, clinical features, and management."
      explanation: >-
        Notes that ACO pathophysiology and definition remain incompletely
        resolved, supporting the cautious CANDIDATE framing.

association_signals:
- source: ICEES
  method: EHR_COHORT_ASSOCIATION
  signal_disorder_a_id: MONDO:0004979
  signal_disorder_b_id: MONDO:0005002
  population: >-
    ICEES KG 8-20-2024, UNC Health primary-ciliary-dyskinesia (PCD) cohort
    (condition-specific base population), patient-level chi-square contingency.
  mapping_notes: >-
    ICEES reports this pair as a positive correlation. Chi-square is not
    multiple-testing corrected and is conditioned on the PCD base population,
    so it corroborates rather than establishes the association. The row quoted
    is the strongest PCD cohort-year (2016).
  directionality: UNKNOWN
  statistics:
    metrics:
    - metric_type: CHI_SQUARE
      metric_value: 168.58533016733276
      p_value: 1.5071340388291068e-38
      notes: >-
        ICEES PCD 2016 cohort co-occurrence of asthma and COPD (N=5688);
        strong, highly significant positive association.
  evidence:
  - reference: ICEES:MONDO_0004979__MONDO_0005002
    supports: SUPPORT
    evidence_source: OTHER
    snippet: "PCD_UNC_patient_2016_v6_binned_deidentified | 168.58533016733276 | 1 | 1.5071340388291068e-38 | 5688"
    explanation: >-
      ICEES UNC EHR cohort shows a strong, highly significant asthma-COPD
      co-occurrence in the 2016 PCD cohort.
Source:GitHub